Evidence map›Paper›PMID 26239408›Full record

ArticleTAG. Theoretical and applied genetics. Theoretische und angewandte Genetik2015

The genetic basis of flecking and its relationship to disease resistance in the IBM maize mapping population.

Vijay Vontimitta, Bode A Olukolu, Bryan W Penning, Gurmukh Johal, P J Balint-Kurti

Abstract read
PubMed Publisher
In one paragraph

Article in TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.5field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 8 citations in OpenAlex.

  1. Quantitative trait locus analysis of gray leaf spot resistance in the maize IBM Syn10 DH population.TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik · 2024
    Article
  2. Review
  3. Article
  4. Article
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 3 institutions in 1 country.

Vijay VontimittaBotany and Plant Pathology, Purdue University, Lilly Hall, West Lafayette, IN, 47907-2054, USA.
Bode A OlukoluDepartment of Plant Pathology, NC State University, 2574 Thomas Hall, Raleigh, NC, 27695-7616, USA.
Bryan W PenningDepartment of Biological Sciences, Purdue University, West Lafayette, IN, 47907, USA.
Gurmukh JohalBotany and Plant Pathology, Purdue University, Lilly Hall, West Lafayette, IN, 47907-2054, USA.
P J Balint-KurtiDepartment of Plant Pathology, NC State University, 2574 Thomas Hall, Raleigh, NC, 27695-7616, USA. peter_balintkurti@ncsu.edu.
Purdue University West Lafayette · USAgricultural Research Service · USNorth Carolina State University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

key messageIn this paper, we determine the genetic architecture controlling leaf flecking in maize and investigate its relationship to disease resistance and the defense response. Flecking is defined as a mild, often environmentally dependent lesion phenotype observed on the leaves of several commonly used maize inbred lines. Anecdotal evidence suggests a link between flecking and enhanced broad-spectrum disease resistance. Neither the genetic basis underlying flecking nor its possible relationship to disease resistance has been systematically evaluated. The commonly used maize inbred Mo17 has a mild flecking phenotype. The IBM-advanced intercross mapping population, derived from a cross between Mo17 and another commonly used inbred B73, has been used for mapping a number of traits in maize including several related to disease resistance. In this study, flecking was assessed in the IBM population over 6 environments. Several quantitative trait loci for flecking were identified, with the strongest one located on chromosome 6. Low but moderately significant correlations were observed between stronger flecking and higher disease resistance with respect to two diseases, southern leaf blight and northern leaf blight and between stronger flecking and a stronger defense response.

Indexed as

Chromosome MappingQuantitative Trait LociCrosses, GeneticDisease ResistanceDNA, PlantPhenotypePlant DiseasesPlant LeavesZea maysDNA, Plant

Identifiers

PMID26239408
OpenAlexW1030739411

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.