Evidence map›Paper›PMID 26179279›Full record

ArticleJournal of molecular neuroscience : MN2015

Effect of Homocysteine on Voltage-Gated Sodium Channel Currents in Primary Cultured Rat Caudate Nucleus Neurons and Its Modulation by 2-Arachidonylglycerol.

Ziliang Zou, Yongli Lu, Manman Dong, Hongwei Yang

Abstract read
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In one paragraph

Article in Journal of molecular neuroscience : MN, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.3field-weighted citation impact, top 48% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 2 citations in OpenAlex.

  1. Pathophysiology of homocysteine: insights into ion channel dysfunction.Pflugers Archiv : European journal of physiology · 2026
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Ziliang ZouDepartment of Physiology and Pathophysiology, College of Medical Sciences, China Three Gorges University, 8 University Road, 443002, Yichang, Hubei,, People's Republic of China.
Yongli LuDepartment of Physiology and Pathophysiology, College of Medical Sciences, China Three Gorges University, 8 University Road, 443002, Yichang, Hubei,, People's Republic of China.
Manman DongDepartment of Physiology and Pathophysiology, College of Medical Sciences, China Three Gorges University, 8 University Road, 443002, Yichang, Hubei,, People's Republic of China.
Hongwei YangDepartment of Physiology and Pathophysiology, College of Medical Sciences, China Three Gorges University, 8 University Road, 443002, Yichang, Hubei,, People's Republic of China. yhw0224@yahoo.com.
China Three Gorges University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Homocysteine (Hcy) is an important risk factor for Alzheimer's disease (AD) and other neurodegenerative diseases. Caudate nucleus (CN), the largest nucleus in the brain, is also implicated in many neurological disorders. 2-Arachidonoylglycerol (2-AG), the most abundant endogenous cannabinoid, has been shown to exhibit neuroprotective effects from many stimuli in the central nervous system (CNS). Furthermore, it has been reported that voltage-gated sodium channels (VGSCs) are the common targets of many neuronal damages and drugs. However, it is still not clear whether VGSCs are involved in the neurotoxicity of Hcy and the neuroprotective effect of 2-AG in CN neurons. In the present study, whole-cell patch clamp recording was used to invest the action of Hcy on sodium currents in primary cultured rat CN neurons and its modulation by 2-AG. The results showed that in cultured CN neurons, pathological concentration of Hcy (100 μM) significantly increased the voltage-gated sodium currents (I(Na)) and produced a hyperpolarizing shift in the activation-voltage curve of I(Na). The further data demonstrated 2-AG is capable of suppressing elevation of Hcy-induced increase in I(Na) and hyperpolarizing shift of activation curves most partly through CB1 receptor-dependent way. Our study provides a better understanding of Hcy-associated neurological disorders and suggests the therapeutic potential for 2-AG for the treatment of these diseases.

Indexed as

Action PotentialsAnimalsArachidonic AcidsCannabinoid Receptor AgonistsCaudate NucleusCells, CulturedEndocannabinoidsGlyceridesHomocysteineNeuronsNeuroprotective AgentsRatsRats, Sprague-DawleyVoltage-Gated Sodium ChannelsArachidonic AcidsCannabinoid Receptor AgonistsEndocannabinoidsGlyceridesglyceryl 2-arachidonateHomocysteineNeuroprotective AgentsVoltage-Gated Sodium Channels2-Arachidonoylglycerol (2-AG)Cannabinoid receptorCaudate nucleus (CN)Homocysteine (Hcy)Sodium channelWhole patch clamp

Identifiers

PMID26179279
OpenAlexW859963316

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.