Evidence map›Paper›PMID 26135875›Full record

ArticleJournal of clinical microbiology2015

Development and Validation of Two Screening Assays for the Hepatitis C Virus NS3 Q80K Polymorphism Associated with Reduced Response to Combination Treatment Regimens Containing Simeprevir.

C K S Chui, W W Y Dong, J B Joy, A F Y Poon, W Y Dong, T Mo, C K Woods, C Beatty, H Hew, P R Harrigan and 1 more

Abstract readValidation Study
In one paragraph

Article in Journal of clinical microbiology, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

C K S ChuiBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada.
W W Y DongBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada.
J B JoyBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada.
A F Y PoonBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.
W Y DongBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada.
T MoBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada.
C K WoodsBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada.
C BeattyBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada.
H HewClinical Affairs, Janssen Inc., North York, Ontario, Canada.
P R HarriganBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada prharrigan@cfenet.ubc.ca cbrumme@cfenet.ubc.ca.
C J BrummeBC Centre for Excellence in HIV/AIDS, Vancouver, British Columbia, Canada prharrigan@cfenet.ubc.ca cbrumme@cfenet.ubc.ca.

Funding

Canadian Institutes of Health Research
6 · The paper itself

Abstract

Persons with hepatitis C virus (HCV) genotype 1a (GT1a) infections harboring a baseline Q80K polymorphism in nonstructural protein 3 (NS3) have a reduced virologic response to simeprevir in combination with pegylated interferon-alfa and ribavirin. We aimed to develop, validate, and freely disseminate an NS3 clinical sequencing assay to detect the Q80K polymorphism and potentially other HCV NS3 drug resistance mutations. HCV RNA was extracted from frozen plasma using a NucliSENS easyMAG automated nucleic acid extractor, amplified by nested reverse transcription-PCR, and sequenced using Sanger and/or next-generation (MiSeq) methods. Sanger chromatograms were analyzed using in-house software (RECall), and nucleotide mixtures were called automatically. MiSeq reads were iteratively mapped to the H77 reference genome, and consensus NS3 sequences were generated with nucleotides present at >20% called as mixtures. The accuracy, precision, and sensitivity for detecting the Q80K polymorphism were assessed in 70 samples previously sequenced by an external laboratory. A comparison of the sequences generated by the Sanger and MiSeq methods with those determined by an external lab revealed >98.5% nucleotide sequence concordance and zero discordant calls of the Q80K polymorphism. The results were both highly repeatable and reproducible (>99.7% nucleotide concordance and 100% Q80K concordance). The limits of detection (>2 and ∼5 log10 IU/ml for the Sanger and MiSeq assays, respectively) are sufficiently low to allow genotyping in nearly all chronically infected treatment-naive persons. No systematic bias in the under- or overamplification of minority variants was observed. Coinfection with other viruses (e.g., HIV and hepatitis B virus [HBV]) did not affect the assay results. The two independent HCV NS3 sequencing assays with the automated analysis procedures described here are useful tools to screen for the Q80K polymorphism and other HCV protease inhibitor drug resistance mutations.

Indexed as

Mutation, MissenseAntiviral AgentsDEAD-box RNA HelicasesDrug ResistanceGenotyping TechniquesHepacivirusHepatitis C, ChronicHumansMass ScreeningMutant ProteinsNucleoside-TriphosphataseReproducibility of ResultsSensitivity and SpecificitySerine EndopeptidasesSimeprevirViral Nonstructural ProteinsAntiviral AgentsDEAD-box RNA HelicasesMutant ProteinsNS3 protein, hepatitis C virusNucleoside-TriphosphataseSerine EndopeptidasesSimeprevirViral Nonstructural ProteinsViral Proteases

Identifiers

PMID26135875
PMCPMC4540893

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.