Evidence map›Paper›PMID 26113176›Full record

ArticleCancer gene therapy2015

Upregulated expression of NKG2D and its ligands give potential therapeutic targets for patients with thymoma.

X Y Xuan, J F Zhang, G M Hu, Q R Li, P P Liu, Y Du

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Article in Cancer gene therapy, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
0.7field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 8 citations in OpenAlex.

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4 · The record

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5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

X Y XuanDepartment of Immunology and Microbiology, Basic Medical College, Zhengzhou University, Zhengzhou, China.
J F ZhangDepartment of Laboratory, The Fifth Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
G M HuDepartment of Pathology, The Second Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Q R LiDepartment of Immunology and Microbiology, Basic Medical College, Zhengzhou University, Zhengzhou, China.
P P LiuDepartment of Immunology and Microbiology, Basic Medical College, Zhengzhou University, Zhengzhou, China.
Y DuDepartment of Immunology and Microbiology, Basic Medical College, Zhengzhou University, Zhengzhou, China.
Zhengzhou University · CNFifth Affiliated Hospital of Zhengzhou University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The activating receptor NKG2D (natural killer group 2, member D) of natural killer (NK) cells promotes tumor immune surveillance by targeting ligands selectively induced on cancer cells, and thus having an important role in antitumor immune response. Because these ligands are not widely expressed on healthy adult tissue, NKG2D ligands may present as useful target for immunotherapeutic approaches in cancer. In this study, to elucidate the role of NKG2D-NKG2D ligand interaction in thymoma tissues and to evaluate the potential role of NKG2D ligands as therapeutic target for thymoma, we examined the expression of NKG2D and its specific ligands: MICA (major histocompatibility complex class I chain-related protein A), MICB (major histocompatibility complex class I chain-related protein B) and ULBP (UL16-binding protein) in 36 thymomas (6 subtype A, 6 subtype AB, 8 subtype B1, 5 subtype B2, 6 subtype B3 and 5 subtype C), 15 thymic atrophy and 8 thymic hyperplasia by immunohistochemistry and reverse transcription-real-time-PCR methods. We demonstrated that both mRNA and protein levels of NKG2D, MICA, MICB and ULBP were upregulated in six types of thymomas compared with those in atrophic thymus or proliferating thymus. Furthermore, the NKG2D ligands were found to be frequently coexpressed on thymoma cells. Furthermore, the expression of MICA, MICB and ULBP in subtype C was higher compared with those in subtype A, AB, B1, B2 and B3. Thus, we concluded that high expressions of NKG2D, MICA, MICB and ULBP1 were shown in patients with thymoma, and this may enhance the recognition function of NK cells to eliminate tumor cells. MICA, MICB and ULBP presented an attractive target for thymoma therapy. The abnormal expression of NKG2D, MICA, MICB and ULBP1 can provide us with evidence of the occurrence of thymoma and could also be used as a target in the treatment of thymoma.

Indexed as

Gene ExpressionGPI-Linked ProteinsHistocompatibility Antigens Class IHumansIntracellular Signaling Peptides and ProteinsNK Cell Lectin-Like Receptor Subfamily KThymomaThymus GlandThymus NeoplasmsUp-RegulationGPI-Linked ProteinsHistocompatibility Antigens Class IIntracellular Signaling Peptides and ProteinsKLRK1 protein, humanMHC class I-related chain AMICB antigenNK Cell Lectin-Like Receptor Subfamily KULBP1 protein, human

Identifiers

PMID26113176
OpenAlexW1546458351

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.