Evidence map›Paper›PMID 26011537›Full record

ArticlePloS one2015

Mitochondrial mutations in subjects with psychiatric disorders.

Adolfo Sequeira, Brandi Rollins, Christophe Magnan, Mannis van Oven, Pierre Baldi, Richard M Myers, Jack D Barchas, Alan F Schatzberg, Stanley J Watson, Huda Akil and 2 more

Abstract read
In one paragraph

Article in PloS one, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed, 2 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Pooled it
  2. Pooled it
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  5. Review
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  13. Review
  14. Article
  15. Article
  16. Genetics Factors in Major Depression Disease.Frontiers in psychiatry · 2018
    Review
  17. Review
  18. Article
  19. Article
  20. Upstream Pathways Controlling Mitochondrial Function in Major Psychosis: A Focus on Bipolar Disorder.Canadian journal of psychiatry. Revue canadienne de psychiatrie · 2016
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Adolfo SequeiraFunctional Genomics Laboratory, Department of Psychiatry & Human Behavior, University of California Irvine, Irvine, California, United States of America.
Brandi RollinsFunctional Genomics Laboratory, Department of Psychiatry & Human Behavior, University of California Irvine, Irvine, California, United States of America.
Christophe MagnanSchool of Information and Computer Sciences (ICS), Institute for Genomics and Bioinformatics (IGB), University of California Irvine, Irvine, California, United States of America.
Mannis van OvenDepartment of Forensic Molecular Biology, Erasmus MC, University Medical Center Rotterdam, Rotterdam, the Netherlands.
Pierre BaldiSchool of Information and Computer Sciences (ICS), Institute for Genomics and Bioinformatics (IGB), University of California Irvine, Irvine, California, United States of America.
Richard M MyersHudsonAlpha Institute for Biotechnology, Huntsville, Alabama, United States of America.
Jack D BarchasDepartment of Psychiatry, Weill Cornell Medical College, New York, New York, United States of America.
Alan F SchatzbergDepartment of Psychiatry & Behavioral Sciences, Stanford University, Palo Alto, California, United States of America.
Stanley J WatsonMolecular and Behavioral Neurosciences Institute, University of Michigan, Ann Arbor, Michigan, United States of America.
Huda AkilMolecular and Behavioral Neurosciences Institute, University of Michigan, Ann Arbor, Michigan, United States of America.
William E BunneyDepartment of Psychiatry & Human Behavior, University of California Irvine, Irvine, California, United States of America.
Marquis P VawterFunctional Genomics Laboratory, Department of Psychiatry & Human Behavior, University of California Irvine, Irvine, California, United States of America.

Funding

Mitochondrial Dysfunction In SchizophreniaR01MH085801 · NIMH · UNIVERSITY OF CALIFORNIA-IRVINE · PI VAWTER, MARQUIS PHILIP · 2009 to 2020
$5.0M
Metallothioneins and polyamines in major depression and suicideR01MH097082 · NIMH · UNIVERSITY OF CALIFORNIA-IRVINE · PI SEQUEIRA, ADOLFO · 2012 to 2015
$1.5M
Mitochondrial Deletions in Mood DisordersR21MH099440 · NIMH · UNIVERSITY OF CALIFORNIA-IRVINE · PI VAWTER, MARQUIS PHILIP · 2013 to 2014
$424k
NIMH NIH HHS MH099440NIMH NIH HHS R01 MH085801NIMH NIH HHS R01MH085801NIMH NIH HHS R01 MH097082NIMH NIH HHS R01MH097082NIMH NIH HHS R21 MH099440
6 · The paper itself

Abstract

A considerable body of evidence supports the role of mitochondrial dysfunction in psychiatric disorders and mitochondrial DNA (mtDNA) mutations are known to alter brain energy metabolism, neurotransmission, and cause neurodegenerative disorders. Genetic studies focusing on common nuclear genome variants associated with these disorders have produced genome wide significant results but those studies have not directly studied mtDNA variants. The purpose of this study is to investigate, using next generation sequencing, the involvement of mtDNA variation in bipolar disorder, schizophrenia, major depressive disorder, and methamphetamine use. MtDNA extracted from multiple brain regions and blood were sequenced (121 mtDNA samples with an average of 8,800x coverage) and compared to an electronic database containing 26,850 mtDNA genomes. We confirmed novel and rare variants, and confirmed next generation sequencing error hotspots by traditional sequencing and genotyping methods. We observed a significant increase of non-synonymous mutations found in individuals with schizophrenia. Novel and rare non-synonymous mutations were found in psychiatric cases in mtDNA genes: ND6, ATP6, CYTB, and ND2. We also observed mtDNA heteroplasmy in brain at a locus previously associated with schizophrenia (T16519C). Large differences in heteroplasmy levels across brain regions within subjects suggest that somatic mutations accumulate differentially in brain regions. Finally, multiplasmy, a heteroplasmic measure of repeat length, was observed in brain from selective cases at a higher frequency than controls. These results offer support for increased rates of mtDNA substitutions in schizophrenia shown in our prior results. The variable levels of heteroplasmic/multiplasmic somatic mutations that occur in brain may be indicators of genetic instability in mtDNA.

Indexed as

AdultCase-Control StudiesDNA, MitochondrialDNA Mutational AnalysisElectrophoresis, Agar GelFemaleGenetic LociHumansMaleMental DisordersMiddle AgedMolecular Sequence DataMutationPrefrontal CortexDNA, Mitochondrial

Identifiers

PMID26011537
PMCPMC4444211

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.