ArticleScientific reports2015
Negative regulatory roles of DE-ETIOLATED1 in flowering time in Arabidopsis.
Article in Scientific reports, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
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Who cites it
10 citing papers in PubMed.
- High-temperature-induced FKF1 accumulation promotes flowering through the dispersion of GI and degradation of SVP.Nature plants · 2025Article
- Identification of genetic and environmental factors influencing aerial root traits that support biological nitrogen fixation in sorghum.G3 (Bethesda, Md.) · 2024Article
- Mutations inFrontiers in plant science · 2022Article
- Light and Abscisic Acid Coordinately Regulate Greening of Seedlings.Plant physiology · 2020Article
- Transcriptome-wide effect of DE-ETIOLATED1 (DET1) suppression in embryogenic callus of Carica papaya.Journal of plant research · 2019Article
- A Novel Loop: Mutual Regulation Between Epigenetic Modification and the Circadian Clock.Frontiers in plant science · 2019Review
- The De-Etiolated 1 Homolog of Arabidopsis Modulates the ABA Signaling Pathway and ABA Biosynthesis in Rice.Plant physiology · 2016Article
- LATE ELONGATED HYPOCOTYL regulates photoperiodic flowering via the circadian clock in Arabidopsis.BMC plant biology · 2016Article
- The role of COP1 in repression of photoperiodic flowering.F1000Research · 2016Review
- CONSTITUTIVE PHOTOMORPHOGENIC 10 (COP10) Contributes to Floral Repression under Non-Inductive Short Days in Arabidopsis.International journal of molecular sciences · 2015Article
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7 authors.
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Abstract
Arabidopsis flowers early under long days (LD) and late under short days (SD). The repressor of photomorphogenesis DE-ETIOLATED1 (DET1) delays flowering; det1-1 mutants flower early, especially under SD, but the molecular mechanism of DET1 regulation remains unknown. Here we examine the regulatory function of DET1 in repression of flowering. Under SD, the det1-1 mutation causes daytime expression of FKF1 and CO; however, their altered expression has only a small effect on early flowering in det1-1 mutants. Notably, DET1 interacts with GI and binding of GI to the FT promoter increases in det1-1 mutants, suggesting that DET1 mainly restricts GI function, directly promoting FT expression independent of CO expression. Moreover, DET1 interacts with MSI4/FVE, which epigenetically inhibits FLC expression, indicating that the lack of FLC expression in det1-1 mutants likely involves altered histone modifications at the FLC locus. These data demonstrate that DET1 acts in both photoperiod and autonomous pathways to inhibit expression of FT and SOC1. Consistent with this, the early flowering of det1-1 mutants disappears completely in the ft-1 soc1-2 double mutant background. Thus, we propose that DET1 is a strong repressor of flowering and has a pivotal role in maintaining photoperiod sensitivity in the regulation of flowering time.
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