Evidence map›Paper›PMID 25904780›Full record

ArticleThe Journal of neuroscience : the official journal of the Society for Neuroscience2015

A human monoclonal IgG that binds aβ assemblies and diverse amyloids exhibits anti-amyloid activities in vitro and in vivo.

Yona Levites, Brian O'Nuallain, Rama Devudu Puligedda, Tomas Ondrejcak, Sharad P Adekar, Cindy Chen, Pedro E Cruz, Awilda M Rosario, Sallie Macy, Alexandra J Mably and 7 more

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of neuroscience : the official journal of the Society for Neuroscience, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
1.3field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 22 citations in OpenAlex.

  1. Article
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  7. Gastrointestinal biofilms in health and disease.Nature reviews. Gastroenterology & hepatology · 2021
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

17 authors at 7 institutions in 3 countries.

Yona LevitesDepartment of Neuroscience, Center for Translational Research in Neurodegenerative Disease, College of Medicine, University of Florida, Gainesville, Florida 32610, dessains@mlhs.org levites.yona@ufl.edu.
Brian O'NuallainThe Laboratory of Neurodegenerative Research, Brigham and Women's Hospital, Harvard Institutes of Medicine, Boston, Massachusetts 02115.
Rama Devudu PuligeddaLankenau Institute for Medical Research, Wynnewood, Pennsylvania 19096.
Tomas OndrejcakTrinity College Institute of Neuroscience and Department of Pharmacology and Therapeutics, Trinity College, Dublin 2, Ireland.
Sharad P AdekarLankenau Institute for Medical Research, Wynnewood, Pennsylvania 19096.
Cindy ChenLankenau Institute for Medical Research, Wynnewood, Pennsylvania 19096.ORCID http://orcid.org/0000-0001-8796-7983
Pedro E CruzDepartment of Neuroscience, Center for Translational Research in Neurodegenerative Disease, College of Medicine, University of Florida, Gainesville, Florida 32610.
Awilda M RosarioDepartment of Neuroscience, Center for Translational Research in Neurodegenerative Disease, College of Medicine, University of Florida, Gainesville, Florida 32610.
Sallie MacyUniversity of Tennessee Graduate School of Medicine, Knoxville, Tennessee 37920.
Alexandra J MablyThe Laboratory of Neurodegenerative Research, Brigham and Women's Hospital, Harvard Institutes of Medicine, Boston, Massachusetts 02115.
Dominic M WalshThe Laboratory of Neurodegenerative Research, Brigham and Women's Hospital, Harvard Institutes of Medicine, Boston, Massachusetts 02115.
Ruben VidalDepartment of Pathology and Laboratory Medicine and the Indiana Alzheimer Disease Center, Indiana University School of Medicine, Indianapolis, Indiana 46202, and.ORCID http://orcid.org/0000-0002-5803-3871
Alan SolomonUniversity of Tennessee Graduate School of Medicine, Knoxville, Tennessee 37920.
Daniel BrownDepartment of Pathology and Laboratory Medicine, Lehigh Valley Health Network, Allentown, Pennsylvania 18103.ORCID http://orcid.org/0000-0002-5822-7975
Michael J RowanTrinity College Institute of Neuroscience and Department of Pharmacology and Therapeutics, Trinity College, Dublin 2, Ireland.
Todd E GoldeDepartment of Neuroscience, Center for Translational Research in Neurodegenerative Disease, College of Medicine, University of Florida, Gainesville, Florida 32610.
Scott K DessainLankenau Institute for Medical Research, Wynnewood, Pennsylvania 19096, dessains@mlhs.org levites.yona@ufl.edu.ORCID http://orcid.org/0000-0002-4331-6799
Lankenau Institute for Medical Research · USUniversity of Florida · USBrigham and Women's Hospital · USTrinity College · CAUniversity of Tennessee at Knoxville · USIndiana University – Purdue University Indianapolis · USLehigh Valley Hospital-Pocono · US

Funding

Immune-Mediated Mechanisms Underlying CNS Abeta ClearanceR01AG018454 · NIA · UNIVERSITY OF FLORIDA · PI GOLDE, TODD E · 2000 to 2018
$5.3M
NIA NIH HHS AG18454NIA NIH HHS R01 AG018454
6 · The paper itself

Abstract

Alzheimer's disease (AD) and familial Danish dementia (FDD) are degenerative neurological diseases characterized by amyloid pathology. Normal human sera contain IgG antibodies that specifically bind diverse preamyloid and amyloid proteins and have shown therapeutic potential in vitro and in vivo. We cloned one of these antibodies, 3H3, from memory B cells of a healthy individual using a hybridoma method. 3H3 is an affinity-matured IgG that binds a pan-amyloid epitope, recognizing both Aβ and λ Ig light chain (LC) amyloids, which are associated with AD and primary amyloidosis, respectively. The pan-amyloid-binding properties of 3H3 were demonstrated using ELISA, immunohistochemical studies, and competition binding assays. Functional studies showed that 3H3 inhibits both Aβ and LC amyloid formation in vitro and abrogates disruption of hippocampal synaptic plasticity by AD-patient-derived soluble Aβ in vivo. A 3H3 single-chain variable fragment (scFv) retained the binding specificity of the 3H3 IgG and, when expressed in the brains of transgenic mice using an adeno-associated virus (AAV) vector, decreased parenchymal Aβ amyloid deposition in TgCRND8 mice and ADan (Danish Amyloid) cerebral amyloid angiopathy in the mouse model of FDD. These data indicate that naturally occurring human IgGs can recognize a conformational, amyloid-specific epitope and have potent anti-amyloid activities, providing a rationale to test their potential as antibody therapeutics for diverse neurological and other amyloid diseases.

Indexed as

AmyloidAmyloid beta-PeptidesAnimalsAntibodies, MonoclonalBrainCataractCerebellar AtaxiaCerebral Amyloid AngiopathyDeafnessDementiaHumansImmunoglobulin GMaleMiceMice, TransgenicRatsAmyloidAmyloid beta-PeptidesAntibodies, MonoclonalImmunoglobulin GAlzheimer's diseaseamyloidamyloid betaanimal studyantibodyIVIG

Identifiers

PMID25904780
PMCPMC4405548
OpenAlexW2029645617

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.