Evidence map›Paper›PMID 25895032›Full record

ArticleOncotarget2015

Reactive stroma component COL6A1 is upregulated in castration-resistant prostate cancer and promotes tumor growth.

Yi-Ping Zhu, Fang-Ning Wan, Yi-Jun Shen, Hong-Kai Wang, Gui-Ming Zhang, Ding-Wei Ye

Open access · diamondAbstract read
In one paragraph

Article in Oncotarget, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
1.0field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 30 citations in OpenAlex.

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  18. Expression of COL6A1 predicts prognosis in cervical cancer patients.American journal of translational research · 2016
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Yi-Ping ZhuDepartment of Urology, Fudan University Shanghai Cancer Center, Fudan University, Shanghai, China.
Fang-Ning WanDepartment of Urology, Fudan University Shanghai Cancer Center, Fudan University, Shanghai, China.
Yi-Jun ShenDepartment of Urology, Fudan University Shanghai Cancer Center, Fudan University, Shanghai, China.
Hong-Kai WangDepartment of Urology, Fudan University Shanghai Cancer Center, Fudan University, Shanghai, China.
Gui-Ming ZhangDepartment of Urology, Fudan University Shanghai Cancer Center, Fudan University, Shanghai, China.
Ding-Wei YeDepartment of Urology, Fudan University Shanghai Cancer Center, Fudan University, Shanghai, China.
Fudan University Shanghai Cancer Center · CNShanghai Medical College of Fudan University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Castration-resistant prostate cancer (CRPC) remains the most critical challenge in the clinical management of prostate cancer (PCa). Reactive stromal changes in PCa are likely involved in the emergence of CRPC. In the present study, we identified a novel oncogene termed COL6A1 which was upregulated in the reactive stroma of CRPC. We established an androgen-independent LNCaP (LNCaP-AI) cell line in steroid-reduced (SR) medium within 2 months. We examined COL6A1 expression with western blot during the LNCaP-AI induction, and studied the function of COL6A1 in vitro and in vivo. Immunohistochemical staining of COL6A1 was performed in ten pairs of androgen-sensitive PCa and CRPC samples. We demonstrated that COL6A1 expression was markedly increased in LNCaP-AI cells and CRPC tissues compared with LNCaP cells and paired androgen-sensitive PCa specimens. In vitro, COL6A1 knockdown resulted in G1-S cell cycle arrest and descended vitality. Overexpression of COL6A1 was associated with accelerated S phase entry and elevated vitality in prostate cancer cells. COL6A1 also promoted tumorigenesis of LNCaP cells in vivo. Taken together, these data suggest an important role of COL6A1 in the molecular etiology of castration-resistant prostate cancer, and support the potential use of COL6A1 in CRPC therapy.

Indexed as

Cell Line, TumorCell ProliferationCollagen Type VIGenes, rasHumansMaleProstatic Neoplasms, Castration-ResistantTranscriptional ActivationTransfectionUp-RegulationCol6a1 protein, humanCollagen Type VIcastration-resistant prostate cancerCOL6A1reactive stromatumorigenesis

Identifiers

PMID25895032
PMCPMC4546481
OpenAlexW1501484691

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.