Evidence map›Paper›PMID 25873838›Full record

ArticleTexas Heart Institute journal2015

Regression of a large congenital hepatic arteriovenous malformation.

Hala Mounir Agha, Rania Zakaria, Fatma Alzahraa Mostafa, Hala Hamza

Open access · bronzeAbstract readCase Reports
In one paragraph

Article in Texas Heart Institute journal, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.7field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 12 citations in OpenAlex.

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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

4 authors.

Hala Mounir Agha
Rania Zakaria
Fatma Alzahraa Mostafa
Hala Hamza

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Congenital hepatic arteriovenous malformations are rarely seen in association with persistent neonatal pulmonary hypertension. We report the case of a full-term female newborn who presented with heart failure and respiratory distress soon after birth. Echocardiographic investigation revealed severe persistent pulmonary hypertension and patent ductus arteriosus. Here we report spontaneous regression in size of both the feeder vessel and the vascular bed of the congenital hepatic arteriovenous malformation. We postulate that our conservative use of oral heart failure therapy, in the form of diuretic agents and captopril, decreased the congestion and diameter of the affected vessels.

Indexed as

Remission, SpontaneousArteriovenous MalformationsCaptoprilDiureticsFemaleFurosemideHeart FailureHepatic VeinsHumansHypertrophyInfant, NewbornLiverPersistent Fetal Circulation SyndromeCaptoprilDiureticsFurosemideArteriovenous malformations/diagnosis/hepatic/therapycaptopril/therapeutic usediagnosis, differentialdiuretics/therapeutic useheart failure/etiology/drug therapyinfant, newbornpulmonary hypertension, persistentremission, spontaneousvenous thrombosis

Identifiers

PMID25873838
PMCPMC4382893
OpenAlexW2123853346

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.