Evidence map›Paper›PMID 25849303›Full record

ArticlePloS one2015

Cdx2 polymorphism affects the activities of vitamin D receptor in human breast cancer cell lines and human breast carcinomas.

Claudio Pulito, Irene Terrenato, Anna Di Benedetto, Etleva Korita, Frauke Goeman, Andrea Sacconi, Francesca Biagioni, Giovanni Blandino, Sabrina Strano, Paola Muti and 2 more

Open access · goldAbstract read
In one paragraph

Article in PloS one, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
2.4field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 27 citations in OpenAlex.

  1. Trial
  2. Article
  3. Article
  4. Oral mucositis: the hidden side of cancer therapy.Journal of experimental & clinical cancer research : CR · 2020
    Review
  5. Current evidence for vitamin D in intestinal function and disease.Experimental biology and medicine (Maywood, N.J.) · 2019
    Review
  6. Vitamin D Receptor Is Necessary for Mitochondrial Function and Cell Health.International journal of molecular sciences · 2018
    Article
  7. Article
  8. Review
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 1 institution in 1 country.

Claudio PulitoMolecular Chemoprevention Unit, Regina Elena National Cancer Institute, Rome, Italy.
Irene TerrenatoDepartment of Epidemiology, Regina Elena National Cancer Institute, Rome, Italy.
Anna Di BenedettoDepartment of Pathology, Regina Elena National Cancer Institute, Rome, Italy.
Etleva KoritaMolecular Chemoprevention Unit, Regina Elena National Cancer Institute, Rome, Italy.
Frauke GoemanTranslational Oncogenomics Unit, Regina Elena National Cancer Institute, Rome, Italy.
Andrea SacconiTranslational Oncogenomics Unit, Regina Elena National Cancer Institute, Rome, Italy.
Francesca BiagioniTranslational Oncogenomics Unit, Regina Elena National Cancer Institute, Rome, Italy.
Giovanni BlandinoTranslational Oncogenomics Unit, Regina Elena National Cancer Institute, Rome, Italy.
Sabrina StranoMolecular Chemoprevention Unit, Regina Elena National Cancer Institute, Rome, Italy.
Paola MutiDepartment of Oncology, Juravinski Cancer Center-McMaster University Hamilton, Hamilton, ON L8V 5C2, Ontario, Canada.
Marcella MottoleseDepartment of Pathology, Regina Elena National Cancer Institute, Rome, Italy.
Elisabetta FalvoLaboratory of Pharmacogenomics (Area Molecular Medicine), Regina Elena National Cancer Institute, Rome, Italy.
McMaster University · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Vitamin D plays a role in cancer development and acts through the vitamin D receptor (VDR). It regulates the action of hormone responsive genes and is involved in cell cycle regulation, differentiation and apoptosis. VDR is a critical component of the vitamin D pathway and different common single nucleotide polymorphisms have been identified. Cdx2 VDR polymorphism can play an important role in breast cancer, modulating the activity of VDR. The objective of this study is to assess the relationship between the Cdx2 VDR polymorphism and the activities of VDR in human breast cancer cell lines and carcinomas breast patients. Cdx2 VDR polymorphism and antiproliferative effects of vitamin D treatment were investigated in a panel of estrogen receptor-positive (MCF7 and T-47D) and estrogen receptor-negative (MDA-MB-231, SUM 159PT, SK-BR-3, BT549, MDA-MB-468, HCC1143, BT20 and HCC1954) human breast cancer cell lines. Furthermore, the potential relationship among Cdx2 VDR polymorphism and a number of biomarkers used in clinical management of breast cancer was assessed in an ad hoc set of breast cancer cases. Vitamin D treatment efficacy was found to be strongly dependent on the Cdx2 VDR status in ER-negative breast cancer cell lines tested. In our series of breast cancer cases, the results indicated that patients with variant homozygote AA were associated with bio-pathological characteristics typical of more aggressive tumours, such as ER negative, HER2 positive and G3. Our results may suggest a potential effect of Cdx2 VDR polymorphism on the efficacy of vitamin D treatment in aggressive breast cancer cells (estrogen receptor negative). These results suggest that Cdx2 polymorphism may be a potential biomarker for vitamin D treatment in breast cancer, independently of the VDR receptor expression.

Indexed as

Polymorphism, GeneticBreast NeoplasmsCDX2 Transcription FactorCell Line, TumorFemaleGene Expression Regulation, NeoplasticHomeodomain ProteinsHumansMCF-7 CellsReceptors, CalcitriolRetrospective StudiesVitamin DCDX2 protein, humanCDX2 Transcription FactorHomeodomain ProteinsReceptors, CalcitriolVDR protein, humanVitamin D

Identifiers

PMID25849303
PMCPMC4388514
OpenAlexW2054663221

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.