Evidence map›Paper›PMID 25826285›Full record

ArticleAutoimmunity2015

Temporal phenotypic features distinguish polarized macrophages in vitro.

David W Melton, Linda M McManus, Jonathan A L Gelfond, Paula K Shireman

Open access · greenAbstract read
In one paragraph

Article in Autoimmunity, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 37 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
37citing papers in PubMed, 1 pooled it
2.2field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

37 citing papers in PubMed, 1 synthesis or guideline pooled it, 64 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 3 institutions in 1 country.

David W MeltonDepartment of Surgery and.
Linda M McManus
Jonathan A L Gelfond
Paula K Shireman
The University of Texas Health Science Center at San Antonio · USLongevity Biotech (United States) · USSouth Texas Veterans Health Care System · US

Funding

TISSUE CULTURE---COREP30CA054174 · NCI · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI Lei Zheng · 1991 to 2026
$59.1M
Institute for Integration of Medicine & Science: A Partnership to Improve HealthUL1TR001120 · NCATS · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI CLARK, ROBERT A, HARGREAVES, KENNETH M · 2013 to 2017
$16.5M
Institute for Integration of Medicine & Science: A Partnership to Improve HealthKL2TR001118 · NCATS · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI CLARK, ROBERT A, DOUGHERTY, DONALD M · 2013 to 2017
$3.4M
CHEMOKINES AND IMMUNE CELLS IN HIND LIMB ISCHEMIAR01HL074236 · NHLBI · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI SHIREMAN, PAULA K · 2003 to 2011
$3.0M
Chemokines and Immune Cells in Hind Limb IschemiaR56HL074236 · NHLBI · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI SHIREMAN, PAULA K · 2007 to 2007
$365k
Improved Muscle Regeneration by Anti-miRNA Alteration in Macrophage PolarizationF30HL110743 · NHLBI · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI MELTON, DAVID · 2011 to 2015
$158k
MicroRNA Regulation of Macrophage Polarization in Muscle RegenerationI01BX001186 · VA · SOUTH TEXAS VETERANS HEALTH CARE SYSTEM · PI SHIREMAN, PAULA K · 2012 to 2015
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BLRD VA I01 BX001186NCATS NIH HHS KL2 TR001118NCATS NIH HHS UL1 TR001120NCI NIH HHS NIH-NCI P30CA054174NCI NIH HHS P30 CA054174NHLBI NIH HHS F30 HL110743NHLBI NIH HHS HL074236NHLBI NIH HHS HL110743NHLBI NIH HHS R01 HL074236NHLBI NIH HHS R56 HL074236
6 · The paper itself

Abstract

Macrophages are important in vascular inflammation and environmental factors influence macrophage plasticity. Macrophage transitions into pro-inflammatory (M1) or anti-inflammatory (M2) states have been defined predominately by measuring cytokines in culture media (CM). However, temporal relationships between cellular and secreted cytokines have not been established. We measured phenotypic markers and cytokines in cellular and CM of murine bone marrow-derived macrophages at multiple time points following stimulation with IFN-γ + LPS (M1), IL-4 (M2a) or IL-10 (M2c). Cytokines/proteins in M1-polarized macrophages exhibited two distinct temporal patterns; an early (0.5-3 h), transient increase in cellular cytokines (GM-CSF, KC-GRO, MIP-2, IP-10 and MIP-1β) and a delayed (3-6 h) response that was more sustained [IL-3, regulated on activation normal T cell expressed and secreted (RANTES), and tissue inhibitor of metalloproteinases 1 (TIMP-1)]. M2a-related cytokine/cell markers (IGF-1, Fizz1 and Ym1) were progressively (3-24 h) increased post-stimulation. In addition, novel patterns were observed. First, and unexpectedly, cellular pro-inflammatory chemokines, MCP-1 and MCP-3 but not MCP-5, were comparably increased in M1 and M2a macrophages. Second, Vegfr1 mRNA was decreased in M1 and increased in M2a macrophages. Finally, VEGF-A was increased in the CM of M1 cultures and strikingly reduced in M2a coinciding with increased Vegfr1 expression, suggesting decreased VEGF-A in M2a CM was secondary to increased soluble VEGFR1. In conclusion, macrophage cytokine production and marker expression were temporally regulated and relative levels compared across polarizing conditions were highly dependent upon the timing and location (cellular versus CM) of the sample collection. For most cytokines, cellular production preceded increases in the CM suggesting that cellular regulatory pathways should be studied within 6 h of stimulation. The divergent polarization-dependent expression of Vegfr1 may be essential to controlling VEGF potentially regulating angiogenesis and inflammatory cell infiltration in the vascular niche. The current study expands the repertoire of cytokines produced by polarized macrophages and provides insights into the dynamic regulation of macrophage polarization and resulting cytokines, proteins and gene expression that influence vascular inflammation.

Indexed as

PhenotypeAnimalsCell DifferentiationChemokine CCL2Chemokine CCL4Chemokine CCL7Chemokine CXCL10Chemokine CXCL2Gene Expression RegulationGranulocyte-Macrophage Colony-Stimulating FactorHumansInterferon-gammaInterleukin-10Interleukin-4LipopolysaccharidesMacrophagesCcl2 protein, mouseCcl7 protein, mouseChemokine CCL2Chemokine CCL4Chemokine CCL7Chemokine CXCL10Chemokine CXCL2Cxcl10 protein, mouseCxcl2 protein, mouseGranulocyte-Macrophage Colony-Stimulating FactorIL10 protein, mouseInterferon-gammaInterleukin-10Interleukin-4LipopolysaccharidesTimp1 protein, mouseTissue Inhibitor of Metalloproteinase-1Vascular Endothelial Growth Factor Avascular endothelial growth factor A, mouseVascular Endothelial Growth Factor Receptor-1Chemokinecytokinemacrophagepolarizationtemporal

Identifiers

PMID25826285
PMCPMC4681525
OpenAlexW2099944930

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.