Evidence map›Paper›PMID 25761880›Full record

ArticleAmerican journal of physiology. Renal physiology2015

Effects of moderate global maternal nutrient reduction on fetal baboon renal mitochondrial gene expression at 0.9 gestation.

Susana P Pereira, Paulo J Oliveira, Ludgero C Tavares, António J Moreno, Laura A Cox, Peter W Nathanielsz, Mark J Nijland

Open access · greenAbstract read
In one paragraph

Article in American journal of physiology. Renal physiology, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
8.0field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 33 citations in OpenAlex.

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  10. Impact of early-life diet on long-term renal health.Molecular and cellular pediatrics · 2020
    Review
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  14. Evolution, kidney development, and chronic kidney disease.Seminars in cell & developmental biology · 2019
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 2 countries.

Susana P PereiraCenter for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal; Department of Life Sciences, School of Sciences and Technology, University of Coimbra, Coimbra, Portugal; Center for Pregnancy and Newborn Research, University of Texas Health Science Center, San Antonio, Texas; and.
Paulo J OliveiraCenter for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal; pauloliv@cnc.uc.pt.
Ludgero C TavaresCenter for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal; Department of Life Sciences, School of Sciences and Technology, University of Coimbra, Coimbra, Portugal;
António J MorenoDepartment of Life Sciences, School of Sciences and Technology, University of Coimbra, Coimbra, Portugal;
Laura A CoxDepartment of Genetics, Texas Biomedical Research Institute, San Antonio, Texas.
Peter W NathanielszCenter for Pregnancy and Newborn Research, University of Texas Health Science Center, San Antonio, Texas; and.
Mark J NijlandCenter for Pregnancy and Newborn Research, University of Texas Health Science Center, San Antonio, Texas; and.
The University of Texas Health Science Center at San Antonio · USUniversity of Coimbra · PTTexas Biomedical Research Institute · US

Funding

The Southwest National Primate Research Center Supplement- Infrastructure improvements of ABSL2 holding areasP51OD011133 · OD · TEXAS BIOMEDICAL RESEARCH INSTITUTE · PI Larry S. Schlesinger · 2012 to 2026
$129.7M
WNV REPLIVAX VACCINE IN NON-HUMAN PRIMATESP51RR013986 · NCRR · TEXAS BIOMEDICAL RESEARCH INSTITUTE · PI DAVIS, MICHAEL DUFF · 1999 to 2011
$79.1M
PARTURITION AND FETAL PRIMATE HYPOTHALAMIC FUNCTIONP01HD021350 · NICHD · UNIVERSITY OF WYOMING · PI NIJLAND, MARK J · 1987 to 2018
$19.9M
MOLECULAR AND BIOCHEMICAL GENETICS LABORATORY RENOVATIONC06RR013556 · NCRR · SOUTHWEST FOUNDATION FOR BIOMEDICAL RES · PI SHADE, ROBERT E · 1998 to 1998
–
NCRR NIH HHS C06 RR013556NCRR NIH HHS P51 RR013986NICHD NIH HHS P01 HD021350NIH HHS P51 OD011133
6 · The paper itself

Abstract

Early life malnutrition results in structural alterations in the kidney, predisposing offspring to later life renal dysfunction. Kidneys of adults who were growth restricted at birth have substantial variations in nephron endowment. Animal models have indicated renal structural and functional consequences in offspring exposed to suboptimal intrauterine nutrition. Mitochondrial bioenergetics play a key role in renal energy metabolism, growth, and function. We hypothesized that moderate maternal nutrient reduction (MNR) would adversely impact fetal renal mitochondrial expression in a well-established nonhuman primate model that produces intrauterine growth reduction at term. Female baboons were fed normal chow diet or 70% of control diet (MNR). Fetal kidneys were harvested at cesarean section at 0.9 gestation (165 days gestation). Human Mitochondrial Energy Metabolism and Human Mitochondria Pathway PCR Arrays were used to analyze mitochondrially relevant mRNA expression. In situ protein content was detected by immunohistochemistry. Despite the smaller overall size, the fetal kidney weight-to-body weight ratio was not affected. We demonstrated fetal sex-specific differential mRNA expression encoding mitochondrial metabolite transport and dynamics proteins. MNR-related differential gene expression was more evident in female fetuses, with 16 transcripts significantly altered, including 14 downregulated and 2 upregulated transcripts. MNR impacted 10 transcripts in male fetuses, with 7 downregulated and 3 upregulated transcripts. The alteration in mRNA levels was accompanied by a decrease in mitochondrial protein cytochrome c oxidase subunit VIc. In conclusion, transcripts encoding fetal renal mitochondrial energy metabolism proteins are nutrition sensitive in a sex-dependent manner. We speculate that these differences lead to decreased mitochondrial fitness that contributes to renal dysfunction in later life.

Indexed as

Gene Expression Regulation, DevelopmentalGestational AgeAnimalsFemaleGenes, MitochondrialKidneyMaternal Nutritional Physiological PhenomenaMitochondriaModels, AnimalPapioPregnancyRNA, MessengerRNA, Mitochondrialmitochondrial messenger RNARNA, MessengerRNA, Mitochondrialfetal programminggene expressionkidneymitochondrianonhuman primate

Identifiers

PMID25761880
PMCPMC4587598
OpenAlexW2165319226

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.