Genome-wide association studies of substance use: considerations regarding populations and phenotypes.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

4 citing papers in PubMed.

1. The frequency ofAnnals of general psychiatry · 2018
   Article
2. Genome-wide meta-analysis identifies a novel susceptibility signal at CACNA2D3 for nicotine dependence.American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics · 2017
   Article
3. Molecular genetic approaches to understanding the comorbidity of psychiatric disorders.Development and psychopathology · 2016
   Review
4. Genetic imaging consortium for addiction medicine: From neuroimaging to genes.Progress in brain research · 2016
   Review

Corrections and comments

• Comment on
  Genome-wide association study of nicotine dependence in American populations: identification of novel risk loci in both African-Americans and European-Americans.2015

Authors and funding

2 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

Indexed as