Evidence map›Paper›PMID 25632307›Full record

ArticleCellular and molecular bioengineering2014

α

Hye-Yeong Kim, Eleni A Skokos, Deborah J Myer, Perez Agaba, Anjelica L Gonzalez

Open access · greenAbstract read
In one paragraph

Article in Cellular and molecular bioengineering, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.5field-weighted citation impact, top 35% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
  4. Targeting Neutrophil βBiomolecules · 2023
    Review
  5. Article
  6. Role of the renin-angiotensin system in NETosis in the coronavirus disease 2019 (COVID-19).Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie · 2022
    Review
  7. Article
  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Hye-Yeong KimDepartment of Biomedical Engineering, Malone Engineering Center 314, Yale University, 55 Prospect Street, New Haven, CT 06511, USA.
Eleni A SkokosDepartment of Biomedical Engineering, Malone Engineering Center 314, Yale University, 55 Prospect Street, New Haven, CT 06511, USA.
Deborah J MyerDepartment of Biomedical Engineering, Malone Engineering Center 314, Yale University, 55 Prospect Street, New Haven, CT 06511, USA.
Perez AgabaDepartment of Biomedical Engineering, Malone Engineering Center 314, Yale University, 55 Prospect Street, New Haven, CT 06511, USA.
Anjelica L GonzalezDepartment of Biomedical Engineering, Malone Engineering Center 314, Yale University, 55 Prospect Street, New Haven, CT 06511, USA.
Yale University · US

Funding

Research Training in AnesthesiaT32GM086287 · NIGMS · YALE UNIVERSITY · PI ROBERT SCHONBERGER · 2009 to 2026
$3.7M
NHLBI NIH HHS L40 HL081996NIGMS NIH HHS T32 GM086287
6 · The paper itself

Abstract

In response to inflammatory stimuli, microvascular endothelial cells become activated, initiating the capture and exit of neutrophils from the blood vessel and into the extravascular extracellular matrix (ECM). In the extravascular space, neutrophils bind to ECM proteins, regulating cellular functions via signaling through adhesion molecules known as integrins. The α

Indexed as

AdhesionExtracellular matrix (ECM)Polyethylene glycol (PEG)Reactive oxygen species (ROS)

Identifiers

PMID25632307
PMCPMC4306468
OpenAlexW2046644999

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.