Evidence map›Paper›PMID 25612797›Full record

ReviewCurrent allergy and asthma reports2015

Maternal influences over offspring allergic responses.

Joan M Cook-Mills

Abstract readReview
In one paragraph

Review in Current allergy and asthma reports, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed.

  1. Association of prenatal vitamin E levels with child asthma and wheeze.Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology · 2024
    Article
  2. [Risk factors for cow's milk protein allergy in infants: a multicenter prospective nested case-control study].Zhongguo dang dai er ke za zhi = Chinese journal of contemporary pediatrics · 2024
    Article
  3. Maternal BMI and allergy in children until 3 years of age (JECS).The journal of allergy and clinical immunology. Global · 2022
    Article
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  14. Perceived Versus Actual Aeroallergen Sensitization in Urban Children.The journal of allergy and clinical immunology. In practice
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Joan M Cook-MillsAllergy-Immunology Division, Northwestern University Feinberg School of Medicine, McGaw-M304, 240 E. Huron, Chicago, IL, 60611, USA, j-cook-mills@northwestern.edu.

Funding

5 -Hydroxytryptophan Regulation of Endothelial Cell Signals for Lung InflammationR01HL111624 · NHLBI · NORTHWESTERN UNIVERSITY AT CHICAGO · PI COOK-MILLS, JOAN M · 2013 to 2016
$1.5M
Forms of Vitamin E Have Opposing Effects on InflammationR01AT004837 · NCCIH · NORTHWESTERN UNIVERSITY AT CHICAGO · PI COOK-MILLS, JOAN M · 2008 to 2011
$1.5M
NCCIH NIH HHS R01 AT004837NHLBI NIH HHS R01 HL111624
6 · The paper itself

Abstract

Asthma occurs as a result of complex interactions of environmental and genetic factors. Clinical studies and animal models of asthma indicate offspring of allergic mothers have increased risk of development of allergies. Environmental factors including stress-induced corticosterone and vitamin E isoforms during pregnancy regulate the risk for offspring development of allergy. In this review, we discuss mechanisms for the development of allergic disease early in life, environmental factors that may impact the development of risk for allergic disease early in life, and how the variation in global prevalence of asthma may be explained, at least in part, by some environmental components.

Indexed as

AnimalsAsthmaDendritic CellsHumansHypersensitivityMother-Child RelationsPrevalenceRisk Factors

Identifiers

PMID25612797
PMCPMC4445968

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.