Evidence map›Paper›PMID 25594248›Full record

ArticleMolecular endocrinology (Baltimore, Md.)2015

Sustained reprogramming of the estrogen response after chronic exposure to endocrine disruptors.

Andrea R Patterson, Xiaokui Mo, Ali Shapiro, Karen E Wernke, Trevor K Archer, Craig J Burd

Open access · bronzeAbstract read
In one paragraph

Article in Molecular endocrinology (Baltimore, Md.), 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
1.2field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 21 citations in OpenAlex.

  1. Review
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  4. Impact ofCells · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Andrea R PattersonDepartment of Molecular Genetics (A.R.P., A.S., K.E.W., C.J.B.), The Ohio State University, Center for Biostatistics (X.M.), Ohio State University Wexner Medical Center, and The Arthur G. James Comprehensive Cancer Center (A.R.P., A.S., C.J.B.), Columbus, Ohio 43210; and Laboratory of Molecular Carcinogenesis (T.K.A.), National Institute of Environmental Health Sciences, Research Triangle Park, North Carolina 27709.
Xiaokui Mo
Ali Shapiro
Karen E Wernke
Trevor K Archer
Craig J Burd
The Ohio State University Wexner Medical Center · USNational Institute of Environmental Health Sciences · USThe Ohio State University · US

Funding

Transcriptional Functions of Nuclear Receptors in Cancer CellsZIAES071006 · NIEHS · NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES · PI ARCHER, TREVOR KEITH · 2009 to 2025
$58.0M
Chromatin Dynamics of Endocrine Disruptor Compounds on Estrogen Receptor FunctionR00ES019918 · NIEHS · OHIO STATE UNIVERSITY · PI BURD, CRAIG J · 2013 to 2015
$729k
NIEHS NIH HHS R00 ES019918NIEHS NIH HHS R00ES019918
6 · The paper itself

Abstract

The pervasive nature of estrogenic industrial and dietary compounds is a growing health concern linked to cancer, obesity, and neurological disorders. Prior analyses of endocrine disruptor action have focused primarily on the short-term consequences of exposure. However, these studies are unlikely to reflect the consequences of constant exposures common to industrialized countries. Here we examined the global effects of long-term endocrine disruption on gene transcription and estrogen signaling. Estrogen-dependent breast cancer cell lines were chronically treated with physiologically relevant levels of bisphenol A or genistein for more than 70 passages. Microarray analysis demonstrated global reprogramming of the transcriptome when compared with a similarly cultured control cell line. Estrogen-responsive targets showed diminished expression in both the presence and absence of estrogen. Estrogen receptor recruitment, H3K4 monomethylation, and deoxyribonuclease accessibility were reduced at nearby response elements. Based on these observations, we investigated the potential of long-term endocrine disruptor exposure to initiate persistent transcriptional reprogramming. Culture of chronically exposed cell lines in the absence of the endocrine disruptors did not reverse many of the signaling defects that accumulated during treatment. Taken together, these data demonstrate that chronic exposure to endocrine disrupting compounds can permanently alter physiological hormone signaling.

Indexed as

Cell Line, TumorEarly Growth Response Protein 3Endocrine DisruptorsEpigenesis, GeneticEstrogensGene Expression Regulation, NeoplasticGenetic LociHumansLigandsReceptors, EstrogenSignal TransductionEarly Growth Response Protein 3EGR3 protein, humanEndocrine DisruptorsEstrogensLigandsReceptors, Estrogen

Identifiers

PMID25594248
PMCPMC4347288
OpenAlexW2159678551

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.