ArticleCongenital anomalies2015
Polygenic expression of teratozoospermia and normal fertility in B10.MOL-TEN1 mouse strain.
Article in Congenital anomalies, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
2 citing papers in PubMed, 2 citations in OpenAlex.
- Genetic mapping of a male factor subfertility locus on mouse chromosome 4.Mammalian genome : official journal of the International Mammalian Genome Society · 2018Article
- A Cascade of epistatic interactions regulating teratozoospermia in mice.Mammalian genome : official journal of the International Mammalian Genome Society · 2015Article
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7 authors at 4 institutions in 2 countries.
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Abstract
Subfertility and infertility are two major reproductive health problems in human and domestic animals. The contribution of the genotype to these conditions is poorly understood. To examine the genetic basis of male subfertility, we analyzed its relationship to sperm morphology in B10.MOL-TEN1 mice, which shows high-frequencies (about 50%) of morphologically abnormal sperm. Drastic histological changes were also found in the testis of the B10.MOL-TEN1. Segregation analysis showed that the abnormal sperm phenotype in B10.MOL-TEN1 was inherited and was predictably controlled by at least three loci. We also found that male fertility of this strain was normal. These findings indicate a complicated relationship between sperm morphology and male subfertility.
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