ArticleClinical and molecular hepatology2014
Reversible splenial lesion on the corpus callosum in nonfulminant hepatitis A presenting as encephalopathy.
Article in Clinical and molecular hepatology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
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Who cites it
7 citing papers in PubMed, 13 citations in OpenAlex.
- Clinical Characteristics of H1N1 Influenza A-Associated Mild Encephalopathy with Reversible Splenial Lesion: 4 Pediatric Cases.Current medical science · 2021Article
- Corpus Callosum Involvement as Extrahepatic Manifestation of Hepatitis E Virus: An Uncommon Entity.Journal of neurosciences in rural practice · 2021Article
- Reversible Splenial Lesion Syndrome with Some Novel Causes and Clinical Manifestations.Internal medicine (Tokyo, Japan) · 2020Observational
- Mild encephalitis/encephalopathy with reversible splenial lesion (MERS) in adults-a case report and literature review.BMC neurology · 2017Review
- Encephalitis with reversible splenial and deep cerebral white matter lesions associated with Epstein-Barr virus infection in adults.Neuropsychiatric disease and treatment · 2017Article
- Reversible splenial lesion syndrome associated with lobar pneumonia: Case report and review of literature.Medicine · 2016Article
- Clinically mild encephalitis/encephalopathy with a reversible splenial lesion caused by methicillin-sensitive Staphylococcus aureus bacteremia with toxic shock syndrome: a case report.BMC infectious diseases · 2016Article
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Authors and funding
7 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Reversible focal lesions on the splenium of the corpus callosum (SCC) have been reported in patients with mild encephalitis/encephalopathy caused by various infectious agents, such as influenza, mumps, adenovirus, Varicella zoster, Escherichia coli, Legionella pneumophila, and Staphylococcus aureus. We report a case of a reversible SCC lesion causing reversible encephalopathy in nonfulminant hepatitis A. A 30-year-old healthy male with dysarthria and fever was admitted to our hospital. After admission his mental status became confused, and so we performed electroencephalography (EEG) and magnetic resonance imaging (MRI) of the brain, which revealed an intensified signal on diffusion-weighted imaging (DWI) at the SCC. His mental status improved 5 days after admission, and the SCC lesion had completely disappeared 15 days after admission.
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