Evidence map›Paper›PMID 25468480›Full record

ReviewTranslational research : the journal of laboratory and clinical medicine2015

Targeting of type I interferon in systemic autoimmune diseases.

Mary K Crow, Mikhail Olferiev, Kyriakos A Kirou

Open access · greenAbstract readReview
In one paragraph

Review in Translational research : the journal of laboratory and clinical medicine, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 61 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
61citing papers in PubMed, 1 pooled it
11.7field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

61 citing papers in PubMed, 1 synthesis or guideline pooled it, 124 citations in OpenAlex.

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1 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Mary K CrowMary Kirkland Center for Lupus Research, Hospital for Special Surgery, New York, NY; Rheumatology Division, Department of Medicine, Weill Cornell Medical College, New York, NY. Electronic address: crowm@hss.edu.
Mikhail OlferievMary Kirkland Center for Lupus Research, Hospital for Special Surgery, New York, NY; Rheumatology Division, Department of Medicine, Weill Cornell Medical College, New York, NY.
Kyriakos A KirouMary Kirkland Center for Lupus Research, Hospital for Special Surgery, New York, NY; Rheumatology Division, Department of Medicine, Weill Cornell Medical College, New York, NY.
Hospital for Special Surgery · USCornell University · US

Funding

Interferon in Systemic Lupus ErythematosusR01AI059893 · NIAID · HOSPITAL FOR SPECIAL SURGERY · PI CROW, MARY K · 2006 to 2010
$2.1M
NIAID NIH HHS AI059893NIAID NIH HHS R01 AI059893
6 · The paper itself

Abstract

Increased blood levels of type I interferon (IFN-I) and expression of a broad signature of gene transcripts that reflect induction by IFN-I are observed in many patients with systemic autoimmune diseases, and that pattern is most striking in systemic lupus erythematosus (SLE). Persistent production of IFN-α, the most abundant subtype measured in these patients, is an important feature of the immunopathogenesis of lupus and has stimulated current efforts to develop and test therapeutics that either block IFN-I or its receptor directly or target components of the IFN-I pathway involved in induction of or response to IFN-I. In this review data from animal models of chronic viral infection, examples of lupus-like syndromes associated with single-gene mutations that impact the IFN-I pathway, and longitudinal studies of patients with lupus are described and support the rationale for therapeutic targeting of the IFN-I pathway. However, the complexity of IFN-I regulation and the diversity of its effects on immune system function suggest that the definitive demonstration of that pathway as a valid and productive therapeutic target will only come from clinical trials of agents tested in patients with systemic autoimmune disease, with patients with lupus likely to be the most informative.

Indexed as

AnimalsAutoimmune DiseasesDisease Models, AnimalHumansInterferon Type ILupus Erythematosus, SystemicLupus NephritisMutationRisk FactorsSignal TransductionTranslational Research, BiomedicalVirus DiseasesInterferon Type I

Identifiers

PMID25468480
PMCPMC4306610
OpenAlexW2059504760

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.