ReviewFrontiers in cellular neuroscience2014
Contributions of the immune system to the pathophysiology of traumatic brain injury - evidence by intravital microscopy.
Review in Frontiers in cellular neuroscience, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.
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Who cites it
21 citing papers in PubMed.
- Concussion: Beyond the Cascade.Cells · 2023Review
- Tailored Therapeutic Doses of Dexmedetomidine in Evolving Neuroinflammation after Traumatic Brain Injury.Neurocritical care · 2022Article
- Microenvironmental Variations After Blood-Brain Barrier Breakdown in Traumatic Brain Injury.Frontiers in molecular neuroscience · 2021Review
- Influence of Organic Solvents on Secondary Brain Damage after Experimental Traumatic Brain Injury.Neurotrauma reports · 2020Article
- Depletion of regulatory T cells increases T cell brain infiltration, reactive astrogliosis, and interferon-γ gene expression in acute experimental traumatic brain injury.Journal of neuroinflammation · 2019Article
- PET Evaluation of Microglial Activation in Non-neurodegenerative Brain Diseases.Current neurology and neuroscience reports · 2019Review
- Bazedoxifene protects cerebral autoregulation after traumatic brain injury and attenuates impairments in blood-brain barrier damage: involvement of anti-inflammatory pathways by blocking MAPK signaling.Inflammation research : official journal of the European Histamine Research Society ... [et al.] · 2019Article
- Inversion of neurovascular coupling after subarachnoid hemorrhage in vivo.Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism · 2017Article
- The Role of Microglia in the Etiology and Evolution of Chronic Traumatic Encephalopathy.Shock (Augusta, Ga.) · 2017Review
- Acute changes in neurovascular reactivity after subarachnoid hemorrhage in vivo.Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism · 2017Article
- Nuclear Factor Erythroid 2-Related Factor 2 (Nrf2) Mediates Neuroprotection in Traumatic Brain Injury at Least in Part by Inactivating Microglia.Medical science monitor : international medical journal of experimental and clinical research · 2016Article
- Changes in Binding of [(123)I]CLINDE, a High-Affinity Translocator Protein 18 kDa (TSPO) Selective Radioligand in a Rat Model of Traumatic Brain Injury.Neuromolecular medicine · 2016Article
- Does enoxaparin interfere with HMGB1 signaling after TBI? A potential mechanism for reduced cerebral edema and neurologic recovery.The journal of trauma and acute care surgery · 2016Article
- Dietary Docosahexaenoic Acid Improves Cognitive Function, Tissue Sparing, and Magnetic Resonance Imaging Indices of Edema and White Matter Injury in the Immature Rat after Traumatic Brain Injury.Journal of neurotrauma · 2016Article
- Early Inflammatory Response following Traumatic Brain Injury in Rabbits Using USPIO- and Gd-Enhanced MRI.BioMed research international · 2016Article
- Dynamic Profiling: Modeling the Dynamics of Inflammation and Predicting Outcomes in Traumatic Brain Injury Patients.Frontiers in pharmacology · 2016Article
- Emerging Roles for the Immune System in Traumatic Brain Injury.Frontiers in immunology · 2016Review
- Apolipoprotein E knockout induced inflammatory responses related to microglia in neonatal mice brain via astrocytes.International journal of clinical and experimental medicine · 2015Article
- Editorial: Mechanisms of neuroinflammation and inflammatory neurodegeneration in acute brain injury.Frontiers in cellular neuroscience · 2015Article
- Article
Corrections and comments
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Traumatic brain injury (TBI) results in immediate brain damage that is caused by the mechanical impact and is non-reversible. This initiates a cascade of delayed processes which cause additional-secondary-brain damage. Among these secondary mechanisms, the inflammatory response is believed to play an important role, mediating actions that can have both protective and detrimental effects on the progression of secondary brain damage. Histological data generated extensive information; however, this is only a snapshot of processes that are, in fact, very dynamic. In contrast, in vivo microscopy provides detailed insight into the temporal and spatial patterns of cellular dynamics. In this review, we aim to summarize data which was generated by in vivo microscopy, specifically investigating the immune response following brain trauma, and its potential effects on secondary brain damage.
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