Trial reportHuman genetics2015
Revisiting heritability accounting for shared environmental effects and maternal inheritance.
Trial report in Human genetics, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
11 citing papers in PubMed.
- Unraveling the Heritability of Ocular Rigidity and Myopia-Related Traits: A Siblings-Based Study.Investigative ophthalmology & visual science · 2026Article
- Heritability and shared environmental effects of brain diseases in 12,040 extended families.NPJ dementia · 2025Article
- Delineating maternal influence in regulation of variance in major economic traits of White Leghorns: Bayesian insights.PloS one · 2024Article
- Social Determinants of Health Factors for Gene-Environment COVID-19 Research: Challenges and Opportunities.Advanced genetics (Hoboken, N.J.) · 2022Article
- Repurposing large health insurance claims data to estimate genetic and environmental contributions in 560 phenotypes.Nature genetics · 2019Article
- Relations of mitochondrial genetic variants to measures of vascular function.Mitochondrion · 2018Article
- TheJournal of the American Society of Nephrology : JASN · 2018Review
- Classification of common human diseases derived from shared genetic and environmental determinants.Nature genetics · 2017Article
- Sequential recruitment of study participants may inflate genetic heritability estimates.Human genetics · 2017Article
- Evaluating the contribution of genetics and familial shared environment to common disease using the UK Biobank.Nature genetics · 2016Article
- Pedigree- and SNP-Associated Genetics and Recent Environment are the Major Contributors to Anthropometric and Cardiometabolic Trait Variation.PLoS genetics · 2016Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
9 authors.
Funding
Abstract
Heritability measures the proportion of phenotypic variation attributable to genetic factors. In addition to a shared nuclear genetic component, a number of additional variance components, such as spousal correlation, sibship, household and maternal effects, may have strong contributions to inter-individual phenotype variation. In humans, the confounding effects of these components on heritability have not been studied thoroughly. We sought to obtain unbiased heritability estimates for complex traits in the presence of multiple variance components and also to estimate the contributions of these variance components to complex traits. We compared regression and variance component methods to estimate heritability in simulations when additional variance components existed. We then revisited heritability for several traits in Framingham Heart Study (FHS) participants. Using simulations, we found that failure to account for or misclassification of necessary variance components yielded biased heritability estimates. The direction and magnitude of the bias varied depending on a variance structure and an estimation method. Using the best fitted models to account for necessary variance components, we found that heritability estimates for most FHS traits were overestimated, ranging from 4 to 47 %, when we compared models that considered necessary variance components to models that only considered familial relationships. Spousal correlation explained 14-36 % of phenotypic variation in several anthropometric and lifestyle traits. Maternal and sibling effects also contributed to phenotypic variation, ranging from 3 to 5 % and 4 to 7 %, respectively, in several anthropometric and metabolic traits. Our findings may explain, in part, the missing heritability for some traits.
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