Evidence map›Paper›PMID 25374807›Full record

ReviewWorld journal of nephrology2014

Nephropathy in dietary hyperoxaluria: A potentially preventable acute or chronic kidney disease.

Robert H Glew, Yijuan Sun, Bruce L Horowitz, Konstantin N Konstantinov, Marc Barry, Joanna R Fair, Larry Massie, Antonios H Tzamaloukas

Open access · greenAbstract readReview
In one paragraph

Review in World journal of nephrology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
4.6field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed, 65 citations in OpenAlex.

  1. Article
  2. Twenty-four-hour urine oxalate and risk of chronic kidney disease.Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association · 2024
    Observational
  3. Review
  4. Review
  5. Review
  6. Article
  7. Oxalate homeostasis.Nature reviews. Nephrology · 2023
    Review
  8. Article
  9. Review
  10. Article
  11. Article
  12. Article
  13. Article
  14. Article
  15. Review
  16. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 1 institution in 1 country.

Robert H GlewRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
Yijuan SunRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
Bruce L HorowitzRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
Konstantin N KonstantinovRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
Marc BarryRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
Joanna R FairRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
Larry MassieRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
Antonios H TzamaloukasRobert H Glew, Department of Surgery, University of New Mexico School of Medicine, Albuquerque, NM 87131, United States.
University of New Mexico · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hyperoxaluria can cause not only nephrolithiasis and nephrocalcinosis, but also renal parenchymal disease histologically characterized by deposition of calcium oxalate crystals throughout the renal parenchyma, profound tubular damage and interstitial inflammation and fibrosis. Hyperoxaluric nephropathy presents clinically as acute or chronic renal failure that may progress to end-stage renal disease (ESRD). This sequence of events, well recognized in the past in primary and enteric hyperoxalurias, has also been documented in a few cases of dietary hyperoxaluria. Estimates of oxalate intake in patients with chronic dietary hyperoxaluria who developed chronic kidney disease or ESRD were comparable to the reported average oxalate content of the diets of certain populations worldwide, thus raising the question whether dietary hyperoxaluria is a primary cause of ESRD in these regions. Studies addressing this question have the potential of improving population health and should be undertaken, alongside ongoing studies which are yielding fresh insights into the mechanisms of intestinal absorption and renal excretion of oxalate, and into the mechanisms of development of oxalate-induced renal parenchymal disease. Novel preventive and therapeutic strategies for treating all types of hyperoxaluria are expected to develop from these studies.

Indexed as

Acute oxalate nephropathyAcute tubular necrosisCalcium oxalate nephrolithiasisChronic oxalate nephropathyDietary hyperoxaluriaInflammasomesInterstitial nephritisNephrocalcinosisOxalate transporters

Identifiers

PMID25374807
PMCPMC4220346
OpenAlexW2098971688

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.