Evidence map›Paper›PMID 25350293›Full record

ArticleJournal of visualized experiments : JoVE2014

Analysis of tubular membrane networks in cardiac myocytes from atria and ventricles.

Eva Wagner, Sören Brandenburg, Tobias Kohl, Stephan E Lehnart

Abstract readVideo-Audio Media
In one paragraph

Article in Journal of visualized experiments : JoVE, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed.

  1. Article
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  9. Differences in Effects of Length-Dependent Regulation of Force and CaInternational journal of molecular sciences · 2023
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  14. Local hyperactivation of L-type CaScientific reports · 2021
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Eva WagnerHeart Research Center Goettingen; Clinic of Cardiology & Pulmonology, University Medical Center Goettingen; German Center for Cardiovascular Research (DZHK) partner site Goettingen.
Sören BrandenburgHeart Research Center Goettingen; Clinic of Cardiology & Pulmonology, University Medical Center Goettingen.
Tobias KohlHeart Research Center Goettingen; Clinic of Cardiology & Pulmonology, University Medical Center Goettingen.
Stephan E LehnartHeart Research Center Goettingen; Clinic of Cardiology & Pulmonology, University Medical Center Goettingen; German Center for Cardiovascular Research (DZHK) partner site Goettingen; BioMET, Center for Biomedical Engineering & Technology, University of Maryland School of Medicine; slehnart@med.uni-goettingen.de.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In cardiac myocytes a complex network of membrane tubules--the transverse-axial tubule system (TATS)--controls deep intracellular signaling functions. While the outer surface membrane and associated TATS membrane components appear to be continuous, there are substantial differences in lipid and protein content. In ventricular myocytes (VMs), certain TATS components are highly abundant contributing to rectilinear tubule networks and regular branching 3D architectures. It is thought that peripheral TATS components propagate action potentials from the cell surface to thousands of remote intracellular sarcoendoplasmic reticulum (SER) membrane contact domains, thereby activating intracellular Ca(2+) release units (CRUs). In contrast to VMs, the organization and functional role of TATS membranes in atrial myocytes (AMs) is significantly different and much less understood. Taken together, quantitative structural characterization of TATS membrane networks in healthy and diseased myocytes is an essential prerequisite towards better understanding of functional plasticity and pathophysiological reorganization. Here, we present a strategic combination of protocols for direct quantitative analysis of TATS membrane networks in living VMs and AMs. For this, we accompany primary cell isolations of mouse VMs and/or AMs with critical quality control steps and direct membrane staining protocols for fluorescence imaging of TATS membranes. Using an optimized workflow for confocal or superresolution TATS image processing, binarized and skeletonized data are generated for quantitative analysis of the TATS network and its components. Unlike previously published indirect regional aggregate image analysis strategies, our protocols enable direct characterization of specific components and derive complex physiological properties of TATS membrane networks in living myocytes with high throughput and open access software tools. In summary, the combined protocol strategy can be readily applied for quantitative TATS network studies during physiological myocyte adaptation or disease changes, comparison of different cardiac or skeletal muscle cell types, phenotyping of transgenic models, and pharmacological or therapeutic interventions.

Indexed as

AnimalsCalcium Channels, L-TypeCell SeparationHeart AtriaHeart VentriclesImage Processing, Computer-AssistedIntracellular MembranesMiceMicroscopy, FluorescenceMyocytes, CardiacRyanodine Receptor Calcium Release ChannelSarcolemmaCACNA1C protein, mouseCalcium Channels, L-Typeryanodine receptor 2. mouseRyanodine Receptor Calcium Release Channel

Identifiers

PMID25350293
PMCPMC4541455

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LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.