Elucidating sodium channel NaV1.5 clustering in cardiac myocytes using super-resolution techniques.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

5 citing papers in PubMed.

1. Sodium channel subpopulations with distinct biophysical properties and subcellular localization enhance cardiac conduction.The Journal of general physiology · 2023
   Article
2. Subcellular trafficking and endocytic recycling of KAmerican journal of physiology. Cell physiology · 2022
   Review
3. Ankyrin-G mediates targeting of both NaeLife · 2020
   Article
4. Trafficking and Function of the Voltage-Gated Sodium Channel β2 Subunit.Biomolecules · 2019
   Review
5. Vinculin variant M94I identified in sudden unexplained nocturnal death syndrome decreases cardiac sodium current.Scientific reports · 2017
   Article

Corrections and comments

• Comment on
  Super-resolution imaging reveals that loss of the C-terminus of connexin43 limits microtubule plus-end capture and NaV1.5 localization at the intercalated disc.2014

Authors and funding

3 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

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