Evidence map›Paper›PMID 25267677›Full record

ArticleAntimicrobial agents and chemotherapy2014

Potency and resistance analysis of hepatitis C virus NS5B polymerase inhibitor BMS-791325 on all major genotypes.

Mengping Liu, Maria Tuttle, Min Gao, Julie A Lemm

Abstract read
In one paragraph

Article in Antimicrobial agents and chemotherapy, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Discovery of Novel 3-Hydroxyquinazoline-2,4(1International journal of molecular sciences · 2022
    Article
  2. Article
  3. Article
  4. Article
  5. Interferon-Free Treatments for Chronic Hepatitis C Genotype 1 Infection.Journal of clinical and translational hepatology · 2016
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Mengping LiuDepartment of Virology, Bristol-Myers Squibb Research and Development, Wallingford, Connecticut, USA.
Maria TuttleDepartment of Virology, Bristol-Myers Squibb Research and Development, Wallingford, Connecticut, USA.
Min GaoDepartment of Virology, Bristol-Myers Squibb Research and Development, Wallingford, Connecticut, USA.
Julie A LemmDepartment of Virology, Bristol-Myers Squibb Research and Development, Wallingford, Connecticut, USA Julie.Lemm@bms.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

BMS-791325 is a hepatitis C virus (HCV) inhibitor binding to the thumb domain of the NS5B RNA-dependent RNA polymerase. BMS-791325 is well characterized in genotype 1 (GT1) and exhibits good inhibitory activity (50% effective concentration [EC50], <10 nM) against hybrid replicons containing patient NS5B sequences from GT3a, -4a, and -5a while potency against GT2 is significantly reduced (J. A. Lemm et al., Antimicrob. Agents Chemother. 58:3485-3495, 2014, doi:http://dx.doi.org/10.1128/AAC.02495-13). BMS-791325 potency against GT6a hybrid replicons is more variable, with two of three hybrid clones having EC50s similar to that for GT1 while a third patient clone was ∼ 10 times less susceptible to BMS-791325. To characterize the resistance profile of BMS-791325 beyond GT1, curing studies were performed across GT1a and -3a to -6a and demonstrated that GT1a has the highest resistance barrier versus BMS-791325 while GT6a has the lowest. Selection of GT3 to -6 NS5B chimeric replicon cells at different concentrations of BMS-791325 revealed substitutions in the thumb domain of NS5B at residues 494 and 495 that conferred different levels of resistance to BMS-791325 but remained susceptible to NS5A or NS3 protease inhibitors. In addition, we demonstrate that the reduced potency of BMS-791325 against one GT6a patient is due to an A494 polymorphism present in ∼ 21% of sequences in the European HCV database. The results from this report suggest that BMS-791325 is a candidate for combination treatment of HCV GT3 to -6 chronic infections, and the resistance profiles identified will provide useful information for future clinical development.

Indexed as

GenotypeAmino Acid SubstitutionBenzazepinesBinding SitesCell Line, TumorDrug Resistance, ViralGene ExpressionHepacivirusHepatocytesHumansIndolesInhibitory Concentration 50Nucleic Acid Synthesis InhibitorsProtease InhibitorsProtein BindingProtein Structure, Tertiary8-cyclohexyl-N-((dimethylamino)sulfonyl)-1,1a,2,12b-tetrahydro-11-methoxy-1a-((3-methyl-3,8-diazabicyclo(3.2.1)oct-8-yl)carbonyl)cycloprop(d)indolo(2,1-a)(2)benzazepine-5-carboxamideBenzazepinesIndolesNS-5 protein, hepatitis C virusNucleic Acid Synthesis InhibitorsProtease InhibitorsRNA-Dependent RNA PolymeraseViral Nonstructural Proteins

Identifiers

PMID25267677
PMCPMC4249524

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.