What Makes You Can also Break You, Part III: Mitochondrial Permeability Transition Pore Formation by an Uncoupling Channel within the C-Subunit Ring of the F1FO ATP Synthase?

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

5 citing papers in PubMed.

1. Physiological roles of the mitochondrial permeability transition pore.Journal of bioenergetics and biomembranes · 2017
   Review
2. Alterations in voltage-sensing of the mitochondrial permeability transition pore in ANT1-deficient cells.Scientific reports · 2016
   Article
3. Cell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthase.Pharmacological research · 2015
   Review
4. F-ATPase of Drosophila melanogaster forms 53-picosiemen (53-pS) channels responsible for mitochondrial Ca2+-induced Ca2+ release.The Journal of biological chemistry · 2015
   Article
5. Mitochondrial permeability transition pore induction is linked to formation of the complex of ATPase C-subunit, polyhydroxybutyrate and inorganic polyphosphate.Cell death discovery
   Article

Corrections and comments

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Authors and funding

2 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

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