ArticleBMJ case reports2014
Hyperinfection strongyloidiasis in renal transplant recipients.
Article in BMJ case reports, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
6 citing papers in PubMed, 21 citations in OpenAlex.
- A comprehensive review of Strongyloides stercoralis infection after solid organ and hematopoietic stem cell transplantation.Clinical transplantation · 2022Review
- Clinico-epidemiological spectrum of strongyloidiasis in India: Review of 166 cases.Journal of family medicine and primary care · 2020Review
- Screening of Strongyloides infection using an ELISA test in transplant candidates.Clinics (Sao Paulo, Brazil) · 2019Review
- Solid Organ Transplant and Parasitic Diseases: A Review of the Clinical Cases in the Last Two Decades.Pathogens (Basel, Switzerland) · 2018Review
- Strongyloidiasis Current Status with Emphasis in Diagnosis and Drug Research.Journal of parasitology research · 2017Review
- Article
Corrections and comments
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Authors and funding
1 author at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Strongyloidiasis is infection caused by the nematode Strongyloides stercoralis. Chronic uncomplicated strongyloidiasis is known to occur in immunocompetent individuals while hyperinfection and dissemination occurs in selective immunosuppressed hosts particularly those on corticosteroid therapy. We report two cases of hyperinfection strongyloidiasis in renal transplant recipients and document endoscopic and pathological changes in the involved small bowel. One patient presented with features of dehydration and malnutrition while another developed ileal obstruction and strangulation, requiring bowel resection. Oesophagogastroduodenoscopy showed erythematous and thickened duodenal mucosal folds. Histopathological examination of duodenal biopsies revealed S. stercoralis worms, larvae and eggs embedded in mucosa and submucosa. Wet mount stool preparation showed filariform larvae of S. stercoralis in both cases. Patients were managed with anthelmintic therapy (ivermectin/albendazole) and concurrent reduction of immunosuppression. Both patients had uneventful recovery. Complicated strongyloidiasis should be suspected in immunocompromised hosts who present with abdominal pain, vomiting and diarrhoea, particularly in endemic areas.
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Registered trials
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