ArticleEnvironmental pollution (Barking, Essex : 1987)2014
Particle pollution in Rio de Janeiro, Brazil: increase and decrease of pro-inflammatory cytokines IL-6 and IL-8 in human lung cells.
Article in Environmental pollution (Barking, Essex : 1987), 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.
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Who cites it
18 citing papers in PubMed.
- Prenatal exposure to air pollution affects autophagy, senescence and remodelling proteins in cord blood.ERJ open research · 2025Article
- Cadmium Exposure Induces Inflammation Through Oxidative Stress-Mediated Activation of the NF-κB Signaling Pathway and Causes Heat Shock Response in a Piglet Testis.Biological trace element research · 2025Article
- Preventive Effects of Vanillic Acid Against Lung Inflammation and Oxidative Stress Induced by Dust Particles in Wistar Rats.Journal of cellular and molecular medicine · 2025Article
- PMToxics · 2024Article
- Causal relationship between particulate matter 2.5 (PMJournal of global health · 2023Article
- Porcine Epidemic Diarrhea Virus and Its nsp14 Suppress ER Stress Induced GRP78.International journal of molecular sciences · 2023Article
- The Effects and Pathogenesis of PM2.5 and Its Components on Chronic Obstructive Pulmonary Disease.International journal of chronic obstructive pulmonary disease · 2023Review
- Increased allergic and asthmatic risks in children residing in industrial areas by surveying the pre-inflammatory (IgE, IL-4 and IL-13) biomarkers.Journal of environmental health science & engineering · 2022Article
- The impact of polar fraction of the fine particulate matter on redox responses in different rat tissues.Environmental science and pollution research international · 2019Article
- Proinflammatory effects of dust storm and thermal inversion particulate matter (PMJournal of environmental health science & engineering · 2019Article
- Chitosan oligosaccharides alleviate PMEnvironmental science and pollution research international · 2018Article
- REDUCTION OF PM2.5 TOXICITY ON HUMAN ALVEOLAR EPITHELIAL CELLS A549 BY TEA POLYPHENOLS.Journal of food biochemistry · 2018Article
- Article
- Human health impact assessment of exposure to particulate matter: an AirQ software modeling.Environmental science and pollution research international · 2017Article
- Serum cytokine levels related to exposure to volatile organic compounds and PMInternational journal of chronic obstructive pulmonary disease · 2017Article
- Selective ATP-Binding Cassette Subfamily C Gene Expression and Proinflammatory Mediators Released by BEAS-2B after PMMediators of inflammation · 2017Article
- Biodiesel from soybean promotes cell proliferation in vitro.Toxicology in vitro : an international journal published in association with BIBRA · 2016Article
- Human bronchial epithelial cells exposed in vitro to diesel exhaust particles exhibit alterations in cell rheology and cytotoxicity associated with decrease in antioxidant defenses and imbalance in pro- and anti-apoptotic gene expression.Environmental science and pollution research international · 2016Article
Corrections and comments
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Authors and funding
7 authors.
Funding
Abstract
Particle pollution from urban and industrialized regions in Rio de Janeiro (RJ), Brazil was analyzed for toxic and pro-inflammatory (cytokines: IL-6, IL-8, IL-10) responses in human bronchial epithelial cells. Trace elements contribution was studied. Airborne particulate matter was collected at: three industrial sites Ind-1 (PM10) and Ind-2a and 2b (PM2.5); Centro urban area (PM10) and two rural sites (PM2.5, PM10). PM10 acetone extracts were toxic and did not elicit cytokine release; aqueous extracts were less toxic and stimulated the release of IL-6 and IL-8. PM2.5 aqueous extracts from Ind-2 decreased the release of IL-6 and IL-8. Zinc concentration was higher at the industrial and rural reference sites (Ref-1-2) although metals were not associated to cytokines changes. These results demonstrate that PM from RJ can either increase or decrease cytokine secretion in vitro while being site specific and time dependent.
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