Evidence map›Paper›PMID 2507482›Full record

ReviewJapanese journal of cancer research : Gann1989

Progression: the terminal stage in carcinogenesis.

H C Pitot

Abstract readReview
In one paragraph

Review in Japanese journal of cancer research : Gann, 1989. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed.

  1. Article
  2. In Search of a Unifying Concept in Human Diseases.Diseases (Basel, Switzerland) · 2021
    Article
  3. Article
  4. Review
  5. Review
  6. Article
  7. Article
  8. Article
  9. Article
  10. Stage-specific gene expression during hepatocarcinogenesis in the rat.Journal of cancer research and clinical oncology · 1996
    Review
  11. Hepatic neoplasia: reflections and ruminations.Virchows Archiv : an international journal of pathology · 1995
    Review
  12. Article
  13. Article
  14. Article
  15. Article
  16. What Can Chemical Carcinogenesis Shed Light on the LNT Hypothesis in Radiation Carcinogenesis?Dose-response : a publication of International Hormesis Society
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

H C PitotMcArdle Laboratory for Cancer Research, Medical School, Department of Oncology, University of Wisconsin, Madison 53706.

Funding

TRANSGENIC MODELS OF DIOXIN CARCINOGENICITY AND AH RECEPTOR FUNCTIONP01CA022484 · NCI · UNIVERSITY OF WISCONSIN-MADISON · PI BRADFIELD, CHRISTOPHER A · 1985 to 2011
$9.2M
STOREROOM AND MATERIALS ACQUISITION LABORATORYP30CA007175 · NCI · UNIVERSITY OF WISCONSIN MADISON · PI DRINKWATER, NORMAN R. · 1985 to 2000
$2.4M
NCI NIH HHS CA-07175NCI NIH HHS CA-22484
6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

AnimalsCarcinogensHumansNeoplasmsPhenotypeProto-OncogenesCarcinogens

Identifiers

PMID2507482
PMCPMC5917813

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.