Evidence map›Paper›PMID 25038776›Full record

ReviewCellular and molecular life sciences : CMLS2014

Mechanisms of endothelial cell migration.

U Ruth Michaelis

Abstract readReview
In one paragraph

Review in Cellular and molecular life sciences : CMLS, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 99 papers.

0numbers the graph read from it
0cells of the map it votes in
99citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

99 citing papers in PubMed.

  1. Review
  2. Article
  3. Article
  4. Review
  5. Article
  6. Sema3dInternational journal of biological sciences · 2026
    Article
  7. Article
  8. Article
  9. FPR1-dependent Pro-inflammatory Ccl4Respiratory research · 2025
    Article
  10. Article
  11. Article
  12. Microbiology spectrum · 2025
    Article
  13. Article
  14. Review
  15. Review
  16. Article
  17. Targeting breast tumor extracellular matrix and stroma utilizing nanoparticles.Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico · 2025
    Review
  18. Review
  19. Article
  20. Article

39 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

U Ruth MichaelisInstitut für Kardiovaskuläre Physiologie, Goethe-Universität, Theodor-Stern-Kai 7, 60590, Frankfurt am Main, Germany, michaelis@vrc.uni-frankfurt.de.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cell migration plays a central role in a variety of physiological and pathological processes during our whole life. Cellular movement is a complex, tightly regulated multistep process. Although the principle mechanisms of migration follow a defined general motility cycle, the cell type and the context of moving influences the detailed mode of migration. Endothelial cells migrate during vasculogenesis and angiogenesis but also in a damaged vessel to restore vessel integrity. Depending on the situation they migrate individually, in chains or sheets and complex signaling, intercellular signals as well as environmental cues modulate the process. Here, the different modes of cell migration, the peculiarities of endothelial cell migration and specific guidance molecules controlling this process will be reviewed.

Indexed as

Cell MovementAnimalsBlood VesselsEndothelial CellsEphrinsHumansNeovascularization, PhysiologicNerve Growth FactorsNetrin-1NeuronsSemaphorinsSignal TransductionTumor Suppressor ProteinsWound HealingEphrinsNerve Growth FactorsNetrin-1SemaphorinsTumor Suppressor Proteins

Identifiers

PMID25038776
PMCPMC11113960

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.