ReviewPulmonary circulation2014
Gas exchange and pulmonary hypertension following acute pulmonary thromboembolism: has the emperor got some new clothes yet?
Review in Pulmonary circulation, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
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Who cites it
7 citing papers in PubMed, 23 citations in OpenAlex.
- Developing a novel deep learning-based model for automatic right ventricular parameters assessment on ctpa in pulmonary embolism.Emergency radiology · 2025Article
- Nocturnal hypoxemia is associated with poor pulmonary hemodynamics in patients with chronic thromboembolic pulmonary disease.Respiratory research · 2025Article
- Pulmonary embolism with patent Foramen Ovale: a distinct clinical entity demanding tailored management and screening.Frontiers in medicine · 2025Article
- Dual-layer dual-energy CT-derived pulmonary perfusion for the differentiation of acute pulmonary embolism and chronic thromboembolic pulmonary hypertension.European radiology · 2024Article
- Pathophysiology and Management of Pulmonary Embolism.The International journal of angiology : official publication of the International College of Angiology, Inc · 2022Article
- Pulmonary artery hemodynamics are associated with duration of nocturnal desaturation but not apnea-hypopnea index.Journal of clinical sleep medicine : JCSM : official publication of the American Academy of Sleep Medicine · 2020Article
- Clinical risk stratification in COVID-19: the need for a revised approach?Pulmonary circulationArticle
Corrections and comments
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Patients present with a wide range of hypoxemia after acute pulmonary thromboembolism (APTE). Recent studies using fluorescent microspheres demonstrated that the scattering of regional blood flows after APTE, created by the embolic obstruction unique in each patient, significantly worsened regional ventilation/perfusion (V/Q) heterogeneity and explained the variability in gas exchange. Furthermore, earlier investigators suggested the roles of released vasoactive mediators in affecting pulmonary hypertension after APTE, but their quantification remained challenging. The latest study reported that mechanical obstruction by clots accounted for most of the increase in pulmonary vascular resistance, but that endothelin-mediated vasoconstriction also persisted at significant level during the early phase.
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Registered trials
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