ArticleSocial psychiatry and psychiatric epidemiology2014
Postnatal risk environments, epigenetics, and psychosis: putting the pieces together.
Article in Social psychiatry and psychiatric epidemiology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed, 5 citations in OpenAlex.
- The Epigenetic Link between Prenatal Adverse Environments and Neurodevelopmental Disorders.Genes · 2017Review
- Novel methods in psychiatric epidemiology.Social psychiatry and psychiatric epidemiology · 2016Article
- Gene-environment interaction in major depression: focus on experience-dependent biological systems.Frontiers in psychiatry · 2015Review
Corrections and comments
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Authors and funding
1 author at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Postnatal environmental factors, such as early life adversity, cannabis use, and social stressors are associated with increased risk for psychotic disorders. Understanding mechanisms that underlie increased psychosis risk is of great importance for the development of novel preventive approaches and early interventions. In a timely review article, Pishva et al. discuss available evidence suggesting that postnatal environmental risk factors contribute to psychotic disorders via epigenetic mechanisms. While the evidence supporting this hypothesis is limited and primarily based on the epigenetic profiling of psychotic patients and animal models, further investigation in this area is warranted and may bring exciting results.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.