Evidence map›Paper›PMID 24914360›Full record

ReviewWorld journal of gastroenterology2014

Helicobacter pylori infection: new pathogenetic and clinical aspects.

Krisztina Hagymási, Zsolt Tulassay

Open access · bronzeAbstract readReview
In one paragraph

Review in World journal of gastroenterology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed, 1 pooled it
6.2field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 1 synthesis or guideline pooled it, 56 citations in OpenAlex.

  1. Pooled it
  2. Review
  3. Gastroenterology and hepatology from bed to bench · 2025
    Article
  4. Construction and evaluation of aFrontiers in immunology · 2025
    Article
  5. Association betweenBMJ open gastroenterology · 2024
    Article
  6. Pangenome Analysis ofInternational journal of genomics · 2024
    Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Ferroptosis and its Role in Gastric Cancer.Frontiers in cell and developmental biology · 2022
    Review
  12. Article
  13. Review
  14. Epigenetic downregulation of MUC17 by H. pylori infection facilitates NF-κB-mediated expression of CEACAM1-3S in human gastric cancer.Gastric cancer : official journal of the International Gastric Cancer Association and the Japanese Gastric Cancer Association · 2019
    Article
  15. Review
  16. Article
  17. Prevalence ofInternational journal of microbiology · 2018
    Article
  18. Host pathogen interactions inWorld journal of gastroenterology · 2017
    Review
  19. Review
  20. Vaccine against Helicobacter pylori: Inevitable approach.World journal of gastroenterology · 2016
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Krisztina HagymásiKrisztina Hagymási, Zsolt Tulassay, 2 Department of Internal Medicine, Semmelweis University, H-1088 Budapest, Hungary.
Zsolt TulassayKrisztina Hagymási, Zsolt Tulassay, 2 Department of Internal Medicine, Semmelweis University, H-1088 Budapest, Hungary.
Semmelweis University · HU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Helicobacter pylori (H. pylori) infects more than half of the world's human population, but only 1% to 3% of infected people consequently develop gastric adenocarcinomas. The clinical outcome of the infection is determined by host genetic predisposition, bacterial virulence factors, and environmental factors. The association between H. pylori infection and chronic active gastritis, peptic ulcer disease, gastric cell carcinoma, and B cell mucosa-associated lymphoid tissue lymphoma has been well established. With the exception of unexplained iron deficiency anemia and idiopathic thrombocytopenic purpura, H. pylori infection has no proven role in extraintestinal diseases. On the other hand, there is data showing that H. pylori infection could be beneficial for some human diseases. The unpredictability of the long-term consequences of H. pylori infection and the economic challenge in eradicating it is why identification of high-risk individuals is crucial.

Indexed as

Cardiovascular DiseasesColorectal NeoplasmsComorbidityGenomic IslandsHelicobacter InfectionsHelicobacter pyloriHematologic DiseasesHumansImmune SystemInsulin ResistancePancreatic NeoplasmsPeptic UlcerRisk FactorsSkin DiseasesStomach NeoplasmsTreatment OutcomeVirulence FactorsExtraintestinal disordersGastroduodenal diseasesHelicobacter pyloriHost factorsVirulence factor

Identifiers

PMID24914360
PMCPMC4047324
OpenAlexW1986962918

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.