Evidence map›Paper›PMID 24859608›Full record

ArticleNeuropharmacology2014

The neurotoxin domoate causes long-lasting inhibition of the kainate receptor GluK5 subunit.

Janet L Fisher

Open access · greenAbstract read
In one paragraph

Article in Neuropharmacology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.3field-weighted citation impact, top 48% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 7 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

Janet L FisherDepartment of Pharmacology, Physiology and Neuroscience, University of South Carolina School of Medicine, Columbia, SC 29208, USA. Electronic address: jfisher@uscmed.sc.edu.
University of South Carolina · US

Funding

Subunit dependent properties of kainate receptorsR01NS065869 · NINDS · UNIVERSITY OF SOUTH CAROLINA AT COLUMBIA · PI FISHER, JANET L, MOTT, DAVID D · 2009 to 2013
$1.5M
NINDS NIH HHS R01 NS065869NINDS NIH HHS R01NS065869
6 · The paper itself

Abstract

Ionotropic glutamate receptors (iGluRs) are responsible for fast excitatory neurotransmission in the mammalian brain, and are critical regulators of neuronal activity and synaptic plasticity. The three main types of iGluRs (AMPA, NMDA, and kainate receptors) are composed of distinct subunit populations. The tetrameric kainate receptors can be assembled from a combination of five different types of subunits (GluK1-GluK5). GluK1-3 subunits are able to produce functional homomeric receptors, while GluK4-5 are obligate heteromers, and must assemble with a GluK1-3 subunit. The neurotoxin domoate is widely used as an agonist at kainate-type receptors because it produces a less desensitizing response compared to glutamate. We have identified an additional, subunit-dependent action of domoate at recombinant kainate receptors. When applied to heteromeric GluK2/K5 receptors, domoate generates a small, long-lasting, tonic current. In addition, brief exposure to domoate inhibits the GluK5 subunit, preventing its activation by other agonists for several minutes. These characteristics are not associated with the GluK1, K2, or K4 subunits and can be prevented by a mutation in GluK5 that reduces agonist binding affinity. The results also show that the domoate-bound, GluK2/K5 heteromeric receptors can be fully activated by agonists acting through the GluK2 subunit, suggesting that the subunits within the tetramer can function independently to open the ion channel, and that the domoate-bound state is not a desensitized or blocked conformation. This study describes new properties associated with domoate action at kainate receptors, and further characterizes the distinct roles played by different subunits in heteromeric receptors.

Indexed as

6-Cyano-7-nitroquinoxaline-2,3-dioneBinding SitesExcitatory Amino Acid AntagonistsGluK2 Kainate ReceptorGlutamic AcidHEK293 CellsHumansKainic AcidKainic Acid ReceptorsMembrane PotentialsMutationNeural InhibitionNeurotoxinsPatch-Clamp TechniquesTransfection6-Cyano-7-nitroquinoxaline-2,3-dionedomoic acidExcitatory Amino Acid AntagonistsGluK2 Kainate ReceptorGlutamic AcidKainic AcidKainic Acid ReceptorsNeurotoxinsDomoic acidElectrophysiologyKainate receptors

Identifiers

PMID24859608
PMCPMC4107164
OpenAlexW2074781465

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.