Evidence map›Paper›PMID 24829898›Full record

ReviewFrontiers in pediatrics2014

The multifactorial origin of respiratory morbidity in patients surviving neonatal repair of esophageal atresia.

Ana Catarina Fragoso, Juan A Tovar

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in pediatrics, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
3.8field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 32 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Review
  5. Article
  6. Development and stem cells of the esophagus.Seminars in cell & developmental biology · 2017
    Review
  7. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 2 countries.

Ana Catarina FragosoINGEMM and Idipaz Research Laboratory, Department of Pediatric Surgery, Hospital Universitario La Paz , Madrid , Spain ; Department of Pediatrics, Universidad Autonoma de Madrid , Madrid , Spain ; Faculty of Medicine, University of Porto , Porto , Portugal.
Juan A TovarINGEMM and Idipaz Research Laboratory, Department of Pediatric Surgery, Hospital Universitario La Paz , Madrid , Spain ; Department of Pediatrics, Universidad Autonoma de Madrid , Madrid , Spain.
Hospital Universitario La Paz · ESUniversidad Autónoma de Madrid · ES

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Esophageal atresia with or without tracheoesophageal fistula (EA ± TEF) occurs in 1 out of every 3000 births. Current survival approaches 95%, and research is therefore focused on morbidity and health-related quality of life issues. Up to 50% of neonates with EA ± TEF have one or more additional malformations including those of the respiratory tract that occur in a relatively high proportion of them and particularly of those with vertebral, anal, cardiac, tracheoesophageal, renal, and limb association. Additionally, a significant proportion of survivors suffer abnormal pulmonary function and chronic respiratory tract disease. The present review summarizes the current knowledge about the nature of these symptoms in patients treated for EA ± TEF, and explores the hypothesis that disturbed development and maturation of the respiratory tract could contribute to their pathogenesis.

Indexed as

developmentesophageal atresiahumanlungrespiratory tract diseaserodent modelstracheoesophageal fistula

Identifiers

PMID24829898
PMCPMC4017156
OpenAlexW2099427814

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.