Platelet CD36 links not only α-granule-derived proteins to thrombus stability but also metabolic and oxidant stress to a prothrombotic phenotype.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

3 citing papers in PubMed.

1. Thrombospondin-1 protects against pathogen-induced lung injury by limiting extracellular matrix proteolysis.JCI insight · 2018
   Article
2. Monitoring storage induced changes in the platelet proteome employing label free quantitative mass spectrometry.Scientific reports · 2017
   Article
3. Deep vein thrombosis is accurately predicted by comprehensive analysis of the levels of microRNA-96 and plasma D-dimer.Experimental and therapeutic medicine · 2016
   Article

Corrections and comments

• Comment on
  Supporting roles of platelet thrombospondin-1 and CD36 in thrombus formation on collagen.2014

Authors and funding

1 author.

Funding

Abstract

PubMed holds no abstract for this paper.

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