Evidence map›Paper›PMID 24800176›Full record

ArticleOncoimmunology2014

NKp44L: A new tool for fighting cancer.

Vincent Vieillard, Florence Baychelier, Patrice Debré

Abstract read
In one paragraph

Article in Oncoimmunology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. Review
  4. Review
  5. Review
  6. Article
  7. Review
  8. MLL5 (KMT2E): structure, function, and clinical relevance.Cellular and molecular life sciences : CMLS · 2017
    Review
  9. Article
  10. Article
  11. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Vincent VieillardSorbonne Université; UPMC, Univ Paris 06; CR7; CIMI-Paris; Paris, France ; INSERM, U1135; CIMI-Paris; Paris, France ; CNRS, ERL8255; CIMI-Paris; Paris, France.
Florence BaychelierSorbonne Université; UPMC, Univ Paris 06; CR7; CIMI-Paris; Paris, France ; INSERM, U1135; CIMI-Paris; Paris, France ; CNRS, ERL8255; CIMI-Paris; Paris, France.
Patrice DebréSorbonne Université; UPMC, Univ Paris 06; CR7; CIMI-Paris; Paris, France ; INSERM, U1135; CIMI-Paris; Paris, France ; CNRS, ERL8255; CIMI-Paris; Paris, France.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

We have recently identified that the ligand of natural cytotoxicity triggering receptor 2 (NCR2, best known as NKp44) is expressed on a large panel of malignant cells. This ligand provides a new tool to investigate how stressed cells are recognized and eliminated by natural killer (NK) cells, and to develop novel immunotherapeutic paradigms against cancer.

Indexed as

immunotherapyMLL5natural cytotoxicity receptorsnatural killer cellsNKp44NKp44Ltumor cells

Identifiers

PMID24800176
PMCPMC4008457

What OpenQuestion holds

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LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.