Evidence map›Paper›PMID 24719676›Full record

ReviewCommunicative & integrative biology2013

Formin' cellular structures: Physiological roles of Diaphanous (Dia) in actin dynamics.

Sven Bogdan, Jörg Schultz, Jörg Grosshans

Open access · goldAbstract readReview
In one paragraph

Review in Communicative & integrative biology, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
2.0field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed, 41 citations in OpenAlex.

  1. Neuronal Actin Remodeling and Its Role in Higher Nervous Activity.International journal of molecular sciences · 2025
    Review
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  10. The complexities of PKCα signaling in cancer.Advances in biological regulation · 2021
    Review
  11. Article
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  13. The advantages of microfluidics to study actin biochemistry and biomechanics.Journal of muscle research and cell motility · 2020
    Review
  14. Article
  15. Review
  16. Article
  17. Review
  18. Article
  19. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 3 institutions in 1 country.

Sven BogdanInstitut für Neurobiologie; Universität Münster; Münster, Germany.
Jörg SchultzBioinformatik, Biozentrum; Universität Würzburg; Würzburg, Germany.
Jörg GrosshansInstitut für Biochemie; Universitätsmedizin; Universität Göttingen; Göttingen, Germany.
University of Göttingen · DEUniversity of Münster · DEUniversity of Würzburg · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Members of the Diaphanous (Dia) protein family are key regulators of fundamental actin driven cellular processes, which are conserved from yeast to humans. Researchers have uncovered diverse physiological roles in cell morphology, cell motility, cell polarity, and cell division, which are involved in shaping cells into tissues and organs. The identification of numerous binding partners led to substantial progress in our understanding of the differential functions of Dia proteins. Genetic approaches and new microscopy techniques allow important new insights into their localization, activity, and molecular principles of regulation.

Indexed as

actincytoskeletondevelopmentDrosophilaforminnucleator

Identifiers

PMID24719676
PMCPMC3977921
OpenAlexW2062627700

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.