ArticleCase reports in anesthesiology2014
Negative pressure pulmonary edema after reversing rocuronium-induced neuromuscular blockade by sugammadex.
Article in Case reports in anesthesiology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
9 citing papers in PubMed.
- Effects of sugammadex and rocuronium on electro-mechanical activity of cardiac myocytes.Korean journal of anesthesiology · 2025Article
- Sugammadex for our little ones: a brief narrative review.Anesthesia and pain medicine · 2024Review
- Article
- Adverse events of sugammadex that occurred in a Korean population.Anesthesia and pain medicine · 2022Article
- Advantages and pitfalls of clinical application of sugammadex.Anesthesia and pain medicine · 2020Review
- Negative pressure pulmonary edema after general anesthesia: A case report and literature review.Medicine · 2019Review
- Diffuse alveolar hemorrhage following sugammadex and remifentanil administration: A case report.Medicine · 2019Article
- Postoperative negative pressure pulmonary edema following repetitive laryngospasm even after reversal of neuromuscular blockade by sugammadex: a case report.Korean journal of anesthesiology · 2017Article
- Sugammadex: watch out for new side effects.Korean journal of anesthesiology · 2016Article
Corrections and comments
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Negative pressure pulmonary edema (NPPE) is a rare complication that accompanies general anesthesia, especially after extubation. We experienced a case of negative pressure pulmonary edema after tracheal extubation following reversal of rocuronium-induced neuromuscular blockade by sugammadex. In this case, the contribution of residual muscular block on the upper airway muscle as well as large inspiratory forces created by the respiratory muscle which has a low response to muscle relaxants, is suspected as the cause.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.