Evidence map›Paper›PMID 24690899›Full record

ArticlePloS one2014

Using linkage analysis to detect gene-gene interaction by stratifying family data on known disease, or disease-associated, alleles.

Barbara Corso, David A Greenberg

Abstract read
In one paragraph

Article in PloS one, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

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2 · The registry

The trial behind it

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3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Barbara CorsoNational Council Research, Neuroscience Institute, Padova, Italy.
David A GreenbergBattelle Center for Mathematical Medicine, Nationwide Children's Hospital, Columbus, Ohio, United States of America; Department of Pediatrics, Wexner Medical Center, Ohio State University, Columbus, Ohio, United States of America.

Funding

RESOLVING HETEROGENEITY IN EPILEPSY WITH GENETIC MARKERSR01NS027941 · NINDS · MOUNT SINAI SCHOOL OF MEDICINE OF NYU · PI GREENBERG, DAVID A. · 1990 to 2010
$9.0M
Mechanisms of Genetic Seizure Susceptibility in Juvenile Myoclonic EpilepsyR01NS061829 · NINDS · RESEARCH INST NATIONWIDE CHILDREN'S HOSP · PI GREENBERG, DAVID A. · 2009 to 2012
$2.0M
Genetic Analysis: Psychiatric and Other Complex DiseasesT32MH065213 · NIMH · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI WEISSMAN, MYRNA M · 2002 to 2011
$1.7M
PSYCHIATRIC GENETICS AND FAMILY STUDIES--ROBUST METHODSR01MH048858 · NIMH · COLUMBIA UNIV NEW YORK MORNINGSIDE · PI HODGE, SUSAN E. · 1992 to 2010
$1.5M
The Role of Genome-Coded ME2 in EpilepsyR21NS070323 · NINDS · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI GREENBERG, DAVID A. · 2009 to 2010
$798k
NIMH NIH HHS MH48858NIMH NIH HHS R01 MH048858NIMH NIH HHS T32 MH065213NIMH NIH HHS T32-MH65213NINDS NIH HHS NS27941NINDS NIH HHS NS61829NINDS NIH HHS NS70323NINDS NIH HHS R01 NS027941NINDS NIH HHS R01 NS061829NINDS NIH HHS R21 NS070323
6 · The paper itself

Abstract

Detecting gene-gene interaction in complex diseases is a major challenge for common disease genetics. Most interaction detection approaches use disease-marker associations and such methods have low power and unknown reliability in real data. We developed and tested a powerful linkage-analysis-based gene-gene interaction detection strategy based on conditioning the family data on a known disease-causing allele or disease-associated marker allele. We computer-generated multipoint linkage data for a disease caused by two epistatically interacting loci (A and B). We examined several two-locus epistatic inheritance models: dominant-dominant, dominant-recessive, recessive-dominant, recessive-recessive. At one of the loci (A), there was a known disease-related allele. We stratified the family data on the presence of this allele, eliminating family members who were without it. This elimination step has the effect of raising the "penetrance" at the second locus (B). We then calculated the lod score at the second locus (B) and compared the pre- and post-stratification lod scores at B. A positive difference indicated interaction. We also examined if it was possible to detect interaction with locus B based on a disease-marker association (instead of an identified disease allele) at locus A. We also tested whether the presence of genetic heterogeneity would generate false positive evidence of interaction. The power to detect interaction for a known disease allele was 60-90%. The probability of false positives, based on heterogeneity, was low. Decreasing linkage disequilibrium between the disease and marker at locus A decreased the likelihood of detecting interaction. The allele frequency of the associated marker made little difference to the power.

Indexed as

AllelesEpistasis, GeneticGenetic Association StudiesGenetic LinkageComputer SimulationGene FrequencyGenetic HeterogeneityGenetic MarkersHumansLod ScoreModels, GeneticGenetic Markers

Identifiers

PMID24690899
PMCPMC3972093

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.