Evidence map›Paper›PMID 24623445›Full record

ReviewCell and tissue research2014

Vascular events in viral hemorrhagic fevers: a comparative study of dengue and hantaviruses.

Anon Srikiatkhachorn, Christina F Spiropoulou

Open access · hybridAbstract readReview
In one paragraph

Review in Cell and tissue research, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
1.6field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 31 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Article
  5. Article
  6. Article
  7. Article
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  11. Kinetics of viral load and cytokines in severe fever with thrombocytopenia syndrome.Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology · 2018
    Article
  12. Article
  13. Review
  14. Review
  15. Is lactate the new panacea for endothelial dysfunction?Critical care (London, England) · 2014
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 1 country.

Anon SrikiatkhachornDepartment of Medicine, University of Massachusetts Medical School, Worcester, MA, USA, anon.srikiatkhachorn@umassmed.edu.
Christina F Spiropoulou
Centers for Disease Control and Prevention · USUniversity of Massachusetts Chan Medical School · US

Funding

VIRAL DETERMINANTS OF SEVERE DENGUE DISEASEP01AI034533 · NIAID · UNIV OF MASSACHUSETTS MED SCH WORCESTER · PI ROTHMAN, ALAN L · 1994 to 2022
$42.0M
NIAID NIH HHS P01 AI034533NIAID NIH HHS P01AI34533
6 · The paper itself

Abstract

Viral hemorrhagic diseases are a group of systemic viral infections with worldwide distribution and are significant causes of global mortality and morbidity. The hallmarks of viral hemorrhagic fevers are plasma leakage, thrombocytopenia, coagulopathy and hemorrhagic manifestations. The molecular mechanisms leading to plasma leakage in viral hemorrhagic fevers are not well understood. A common theme has emerged in which a complex interplay between pathogens, host immune response, and endothelial cells leads to the activation of endothelial cells and perturbation of barrier integrity. In this article, two clinically distinct viral hemorrhagic fevers caused by dengue viruses and hantaviruses are discussed to highlight their similarities and differences that may provide insights into the pathogenesis and therapeutic approach.

Indexed as

AnimalsDengue VirusEndothelial CellsEndothelium, VascularHemorrhagic Fevers, ViralHumansOrthohantavirus

Identifiers

PMID24623445
PMCPMC3972431
OpenAlexW2000726629

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.