Evidence map›Paper›PMID 24602470›Full record

ReviewAdvances in chronic kidney disease2014

Novel biomarkers in glomerular disease.

Yasar Caliskan, Krzysztof Kiryluk

Open access · greenAbstract readReview
In one paragraph

Review in Advances in chronic kidney disease, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed, 2 pooled it
1.4field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 2 syntheses or guidelines pooled it, 32 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Trial
  4. Review
  5. Article
  6. Article
  7. Article
  8. Review
  9. COSMC expression as a predictor of remission in IgA nephropathy.International urology and nephrology · 2023
    Article
  10. Article
  11. Article
  12. Article
  13. Article
  14. Review
  15. Article
  16. Article
  17. Association of OSAHS Hypoxia Indicators with Early Renal Injury and Serum Fibroblast Growth Factor 21 in Obese Type 2 Diabetic Patients.Diabetes therapy : research, treatment and education of diabetes and related disorders · 2019
    Article
  18. Review
  19. Review
  20. The kidney: function, cells and biomarkers.Jornal brasileiro de nefrologia
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 2 countries.

Yasar CaliskanDivision of Nephrology, Department of Internal Medicine, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey and Division of Nephrology, Department of Medicine, College of Physicians and Surgeons, Columbia University, New York, NY; and Division of Nephrology, Department of Medicine, College of Physicians and Surgeons, Columbia University, New York, NY.
Krzysztof KirylukDivision of Nephrology, Department of Internal Medicine, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey and Division of Nephrology, Department of Medicine, College of Physicians and Surgeons, Columbia University, New York, NY; and Division of Nephrology, Department of Medicine, College of Physicians and Surgeons, Columbia University, New York, NY. Electronic address: kk473@columbia.edu.
Istanbul University · TR

Funding

Quantitative Genetics of Defective IgA1 Glycosylation in IgA NephropathyK23DK090207 · NIDDK · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI KIRYLUK, KRZYSZTOF · 2011 to 2014
$729k
NIDDK NIH HHS K23 DK090207NIDDK NIH HHS K23-DK090207
6 · The paper itself

Abstract

Glomerular diseases are major contributors to the global burden of end-stage kidney disease. The clinical course and outcome of these disorders are extremely variable and difficult to predict. The clinical trajectories range from a benign and spontaneously remitting condition to a symptomatic and rapidly progressive disease. The diagnosis is based entirely on the evaluation of kidney biopsy, but this invasive procedure carries multiple risks and often fails to predict the clinical course or responsiveness to treatment. However, more recent advances in genetics and molecular biology have facilitated elucidation of novel pathogenic mechanisms of these disorders. These discoveries fuel the development of novel biomarkers and offer prospects of noninvasive diagnosis and improved prognostication. Our review focuses on the most promising novel biomarkers that have recently emerged for the major types of glomerular diseases, including immunoglobulin A nephropathy, membranous nephropathy, focal segmental glomerulosclerosis, and membranoproliferative glomerulonephritis.

Indexed as

BiomarkersGlomerulonephritisGenetic MarkersGlomerulonephritis, IGAGlomerulonephritis, MembranoproliferativeGlomerulonephritis, MembranousGlomerulosclerosis, Focal SegmentalHumansKidneyBiomarkersGenetic MarkersFocal segmental glomerulosclerosisGlomerular diseaseImmunoglobulin A nephropathyMembranoproliferative glomerulonephritisMembranous nephropathy

Identifiers

PMID24602470
PMCPMC3963477
OpenAlexW1998493174

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.