ArticleInternational journal of hematology2014
Clinical features and underlying causes of cerebral venous thrombosis in Japanese patients.
Article in International journal of hematology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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Who cites it
5 citing papers in PubMed, 19 citations in OpenAlex.
- Microcytic or Iron Deficiency Anemia as a Risk Factor for Cerebral Sinus Venous Thrombosis-Systematic Review and Exploratory Meta-analysis.TH open : companion journal to thrombosis and haemostasis · 2026Article
- Congenital Thrombophilia in Patients With Superior Mesenteric Venous Thrombosis or Portal Vein Thrombosis.Clinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis · 2018Article
- Seizure in cerebral venous and sinus thrombosis.Epilepsia open · 2018Review
- Comparison of three different anti-Xa assays in major orthopedic surgery patients treated with fondaparinux.International journal of hematology · 2016Article
- High frequency of decreased antithrombin level in pregnant women with thrombosis.International journal of hematology · 2015Article
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Authors and funding
8 authors at 1 institution in 1 country.
Funding
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Abstract
The clinical symptoms, causative factors, and prognosis in Japanese patients with cerebral venous thrombosis have not been adequately characterized. The present study describes these features in patients in Japan. Twenty-two patients with cerebral venous thrombosis were retrospectively identified. Diagnosis was confirmed by either digital subtraction angiography, magnetic resonance venography, or contrast-enhanced computed tomography. Demographic data and clinical and radiological features were recorded and analyzed for each patient. Prognosis was evaluated by the modified Rankin scale (mRS) at the time of hospital discharge. The most frequent symptom of cerebral venous thrombosis was headache (59.1 %). Causative factors included congenital thrombophilia (31.8 %), acquired thrombophilia (27.3 %), and iron-deficiency anemia (13.6 %). Of seven patients with congenital thrombophilia, four had mutations in the protein S gene, two had mutations in the protein C gene, and one had mutations in the antithrombin gene. All patients were alive at discharge from hospital. Nineteen of the 22 patients (86.4 %) recovered completely or exhibited only mild residual symptoms (mRS 0-2). However, three patients (13.6 %) had a poor prognosis (mRS 3-5). Cerebral venous thrombosis in Japanese patients is frequently associated with congenital thrombophilia and protein S gene mutation.
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