ArticleHNO2014
[Hearing research news: from the periphery to the center].
Article in HNO, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed, 3 citations in OpenAlex.
- Stochastic Resonance Controlled Upregulation of Internal Noise after Hearing Loss as a Putative Cause of Tinnitus-Related Neuronal Hyperactivity.Frontiers in neuroscience · 2016Article
- Specific synaptopathies diversify brain responses and hearing disorders: you lose the gain from early life.Cell and tissue research · 2015Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
In the clinical setting, a patient's hearing level is usually quantified by determining the hearing threshold in pure tone and speech audiometry. The measurement and analysis of auditory brainstem responses (ABRs) for quantification of hearing impairment go beyond the scope of routine ENT diagnostic practice. In basic scientific studies, ABRs are commonly used to determine hearing thresholds in animal models. In the last few years, analysis of suprathreshold ABRs, particularly ABR wave I suprathreshold amplitudes, have led to the discovery and understanding of previously undetected hearing impairments in noise-induced and age-related hearing loss models. Deafferentation of inner hair cells and auditory nerve fiber degeneration were identified as pathophysiological correlates to the observed reduction in ABR wave I amplitudes. Such reductions in ABR wave 1 amplitudes could also be identified in chronic tinnitus patients with apparently normal hearing. Recent functional magnetic resonance tomography studies in humans have shown that the limbic system can influence the perception of tinnitus by interacting with an endogenous noise-cancelling system. This highlights the importance of psychotherapeutic treatment approaches for chronic tinnitus.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.