Evidence map›Paper›PMID 24534254›Full record

ArticleEpidemiology and infection2014

Clinical and molecular epidemiology of community-onset invasive Staphylococcus aureus infection in New Zealand children.

D A Williamson, S R Ritchie, S A Roberts, G W Coombs, M G Thomas, O Hannaford, M G Baker, D Lennon, J D Fraser

Open access · bronzeAbstract read
In one paragraph

Article in Epidemiology and infection, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed, 1 pooled it
0.7field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 1 synthesis or guideline pooled it, 16 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Article
  4. Article
  5. Article
  6. Investigation of Virulence Genes ofThe Canadian journal of infectious diseases & medical microbiology = Journal canadien des maladies infectieuses et de la microbiologie medicale · 2021
    Article
  7. Article
  8. Article
  9. Staphylococcus aureus colonisation and its relationship with skin and soft tissue infection in New Zealand children.European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology · 2018
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 5 institutions in 2 countries.

D A WilliamsonFaculty of Medical and Health Sciences,University of Auckland,New Zealand.
S R RitchieFaculty of Medical and Health Sciences,University of Auckland,New Zealand.
S A RobertsDepartment of Clinical Microbiology,Auckland District Health Board,New Zealand.
G W CoombsAustralian Collaborating Centre for Enterococcus and Staphylococcus Species (ACCESS) Typing and Research, PathWest Laboratory Medicine, Perth,Australia.
M G ThomasFaculty of Medical and Health Sciences,University of Auckland,New Zealand.
O HannafordDepartment of Statistics, NZIAS,Massey University,Albany,New Zealand.
M G BakerDepartment of Public Health,University of Otago,Wellington,New Zealand.
D LennonFaculty of Medical and Health Sciences,University of Auckland,New Zealand.
J D FraserFaculty of Medical and Health Sciences,University of Auckland,New Zealand.
University of Auckland · NZAuckland District Health Board · NZCurtin University · AUMassey University · NZUniversity of Otago · NZ

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Our aim was to describe the epidemiology and incidence of community-onset invasive S. aureus disease in children presenting to our hospital, and to compare the clonal complexes and virulence genes of S. aureus strains causing invasive and non-invasive disease. The virulence gene repertoire of invasive disease isolates was characterized using DNA microarray and compared with the virulence gene repertoire of non-invasive S. aureus isolates. Over the study period, 163 children had an invasive S. aureus infection. There was no difference in the distribution of clonal complexes or in the prevalence of genes encoding virulence factors between invasive and non-invasive isolates. Future research should include a strong focus on identifying the host and environmental factors that, along with organism virulence factors, are contributing to the patterns of invasive S. aureus disease observed in New Zealand.

Indexed as

AdolescentChildChild, PreschoolCluster AnalysisCommunity-Acquired InfectionsCross-Sectional StudiesGenotypeHumansIncidenceInfantInfant, NewbornMicroarray AnalysisMolecular EpidemiologyMolecular TypingNew ZealandRetrospective StudiesVirulence Factors

Identifiers

PMID24534254
PMCPMC9161226
OpenAlexW2169994654

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.