ReviewClinical neuroradiology2014
Differential diagnosis of white matter lesions: Nonvascular causes-Part II.
Review in Clinical neuroradiology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
7 citing papers in PubMed.
- Novel Imaging Phenotype in Spinal Cerebellar Ataxia Type 8: Symmetrical White Matter Changes without Cerebellar Atrophy.Cerebellum (London, England) · 2026Article
- Pearls & Oy-sters:Neurology · 2024Article
- Effect of cerebral small vessel disease on cognitive impairment in Parkinson's disease.Acta neurologica Belgica · 2023Article
- Chronic flaccid quadriparesis from tract specific myelopathy in neurosyphilis.Spinal cord series and cases · 2023Article
- Characterization of MRI White Matter Signal Abnormalities in the Pediatric Population.Children (Basel, Switzerland) · 2023Article
- Functional Connectivity Alterations in Neuromyelitis Optica Spectrum Disorder : Correlation with Disease Duration and Cognitive Impairment.Clinical neuroradiology · 2020Article
- Magnetic Resonance Imaging and Clinical Features in Acute and Subacute Myelopathies.Clinical neuroradiology · 2017Review
Corrections and comments
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Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The knowledge of characteristic lesion patterns is important in daily practice imaging, as the radiologist increasingly is required to provide precise differential diagnosis despite unspecific clinical symptoms like cognitive impairment and missed elaborated neurological workup. This part II dealing with nonvascular white matter changes of proven cause and diagnostic significance aimed to assist the evaluation of diseases exhibiting lesions exclusively or predominantly located in the white matter. The etiologies commented on are classified as follows: (a) toxic-metabolic, (b) leukodystrophies and mitochondriopathies, (c) infectious, (d) neoplastic, and (e) immune mediated. The respective mode of lesion formation is characterized, and typical radiological findings are displayed. More or less symmetrical lesion patterns on the one hand as well as focal and multifocal ones on the other are to be analyzed with reference to clinical data and knowledge of predilection sites characterizing major disease categories. Complementing spinal cord imaging may be useful not only in acute and relapsing demyelinating diseases but in certain leukodystrophies as well. In neuromyelitis optica (NMO), the detection of a specific antibody and some recently published observations may lead to a new understanding of certain deep white matter lesions occasionally complicating systemic autoimmune disease.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.