ArticlePlant signaling & behavior2013
The Arabidopsis gene ATST4a in not a typical brassinosteroid catabolic gene.
Article in Plant signaling & behavior, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
5 citing papers in PubMed.
- Identification and evolution of the plant sulfotransferase family.BMC genomics · 2025Article
- Sulfate Availability and Hormonal Signaling in the Coordination of Plant Growth and Development.International journal of molecular sciences · 2024Review
- CIRCADIAN CLOCK ASSOCIATED 1 and ATAF2 differentially suppress cytochrome P450-mediated brassinosteroid inactivation.Journal of experimental botany · 2020Article
- Regulation of Brassinosteroid Homeostasis in Higher Plants.Frontiers in plant science · 2020Review
- The multi-protein family of sulfotransferases in plants: composition, occurrence, substrate specificity, and functions.Frontiers in plant science · 2014Review
Corrections and comments
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Authors and funding
2 authors.
Funding
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Abstract
Brassinosteroid (BR) homeostasis is maintained in part by this hormone’s catabolism. The presence of multiple BR-catabolic pathways in Arabidopsis demonstrates the importance of this process in growth and development. Previous biochemical analyses suggest that AT ST4a has BR catalytic activity. We have used both overexpression and loss-of-function genetic approaches to further explore the role of ATST4a in Arabidopsis. Up to 1000-fold overexpression of the ATST4a gene did not result in any characteristic BR-deficient phenotypes. In addition, the T-DNA insertion null mutant atst4a1-1 did not display enhanced seedling hypocotyl growth in the presence or absence of the active BR brassinolide when grown in white light. This lack of hallmark characteristics for BR-inacitivion genes suggests that ATST4a encodes an atypical BR catabolic enzyme.
Indexed as
Identifiers
24494235PMC4091065What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.