Evidence map›Paper›PMID 24453283›Full record

ReviewQJM : monthly journal of the Association of Physicians2014

Vascular remodelling in the pathogenesis of idiopathic pulmonary fibrosis.

S Barratt, A Millar

Open access · bronzeAbstract readReview
In one paragraph

Review in QJM : monthly journal of the Association of Physicians, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 48 papers.

0numbers the graph read from it
0cells of the map it votes in
48citing papers in PubMed
3.4field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

48 citing papers in PubMed, 73 citations in OpenAlex.

  1. Trial
  2. Article
  3. Article
  4. Article
  5. Review
  6. Article
  7. Exploring sarcoidosis via nailfold capillaroscopy: A window into the disease's microvascular landscape.Sarcoidosis, vasculitis, and diffuse lung diseases : official journal of WASOG · 2025
    Article
  8. Article
  9. Observational
  10. Article
  11. Review
  12. Review
  13. Review
  14. Article
  15. Article
  16. Article
  17. Advancements in preclinical human-relevant modeling of pulmonary vasculature on-chip.European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences · 2024
    Review
  18. Article
  19. Review
  20. Vascular Contribution to Lung Repair and Fibrosis.American journal of respiratory cell and molecular biology · 2023
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

S BarrattFrom the Academic Respiratory Unit, School of Clinical Sciences, University of Bristol, Learning and Research Building, Southmead Hospital, Southmead BS10 5NB, UK shaneybarratt@hotmail.com.
A MillarFrom the Academic Respiratory Unit, School of Clinical Sciences, University of Bristol, Learning and Research Building, Southmead Hospital, Southmead BS10 5NB, UK.
Southmead Hospital · GB

Funding

Wellcome Trust
6 · The paper itself

Abstract

Idiopathic pulmonary fibrosis (IPF) is a progressive and irreversible fibrosing interstitial pneumonia of unknown aetiology that usually leads to respiratory failure and death within 5 years of diagnosis. Alveolar epithelial cell injury, disruption of alveolar capillary membrane integrity and abnormal vascular repair and remodelling have all been proposed as possible pathogenic mechanisms. This review summarizes our current knowledge of the abnormalities in vascular remodelling observed in IPF and highlights several of the cytokines thought to play a pathogenic role, which may ultimately prove to be future therapeutic targets.

Indexed as

Angiostatic ProteinsHumansHypertension, PulmonaryIdiopathic Pulmonary FibrosisMicrocirculationNeovascularization, PathologicVascular RemodelingAngiostatic Proteins

Identifiers

PMID24453283
PMCPMC4071293
OpenAlexW2105325938

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.