ArticlePhotosynthesis research1992
Analysis of a herbicide resistant mutant obtained by transformation of the Chlamydomonas chloroplast.
Article in Photosynthesis research, 1992. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed, 11 citations in OpenAlex.
- Silica Hydrogels as Entrapment Material for Microalgae.Polymers · 2022Review
- A robust protocol for site-directed mutagenesis of the D1 protein inChlamydomonas reinhardtii: A PCR-splicedpsbA gene in a plasmid conferring spectinomycin resistance was introduced into apsbA deletion strain.Photosynthesis research · 1994Article
- Construction of a Chlamydomonas reinhardtii mutant with an intronless psbA gene.Plant molecular biology · 1993Article
Corrections and comments
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
A herbicide resistant Chlamydomonas double mutant (I219A264) has been obtained by transforming the psbA deletion mutant FuD7 with a cloned psbA gene fragment containing mutations in codons 219 and 264. Copies from both the recipient (FuD7) genome and the genome carrying the mutated psbA gene persist in the transformant. This stable heteroplasmic state appears to be required for photoautotrophic growth. Comparison of resistance profiles for classical and phenol-type inhibitors of the double mutant and the corresponding single mutants demonstrates independent, additive contributions of both amino acids to herbicide binding. The approach chosen here to modify the psbA gene should be useful in those cases where consequences of psbA gene manipulations are not predictable with respect to inhibitor resistance.
Identifiers
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.