Evidence map›Paper›PMID 24391515›Full record

ArticlePLoS genetics2014

The SPF27 homologue Num1 connects splicing and kinesin 1-dependent cytoplasmic trafficking in Ustilago maydis.

Nikola Kellner, Kai Heimel, Theresa Obhof, Florian Finkernagel, Jörg Kämper

Open access · goldAbstract read
In one paragraph

Article in PLoS genetics, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.0field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 19 citations in OpenAlex.

  1. Article
  2. Deciphering the RNA-binding protein network during endosomal mRNA transport.Proceedings of the National Academy of Sciences of the United States of America · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Nikola KellnerKarlsruhe Institute of Technology, Institute for Applied Biosciences, Department of Genetics, Karlsruhe, Germany.
Kai HeimelKarlsruhe Institute of Technology, Institute for Applied Biosciences, Department of Genetics, Karlsruhe, Germany ; Georg-August-University-Göttingen, Institute for Microbiology and Genetics, Microbial Cell Biology, Göttingen, Germany.
Theresa ObhofKarlsruhe Institute of Technology, Institute for Applied Biosciences, Department of Genetics, Karlsruhe, Germany.
Florian FinkernagelInstitute for Molecular Biology and Tumor Research, Marburg, Germany.
Jörg KämperKarlsruhe Institute of Technology, Institute for Applied Biosciences, Department of Genetics, Karlsruhe, Germany.
Karlsruhe Institute of Technology · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The conserved NineTeen protein complex (NTC) is an integral subunit of the spliceosome and required for intron removal during pre-mRNA splicing. The complex associates with the spliceosome and participates in the regulation of conformational changes of core spliceosomal components, stabilizing RNA-RNA- as well as RNA-protein interactions. In addition, the NTC is involved in cell cycle checkpoint control, response to DNA damage, as well as formation and export of mRNP-particles. We have identified the Num1 protein as the homologue of SPF27, one of NTC core components, in the basidiomycetous fungus Ustilago maydis. Num1 is required for polarized growth of the fungal hyphae, and, in line with the described NTC functions, the num1 mutation affects the cell cycle and cell division. The num1 deletion influences splicing in U. maydis on a global scale, as RNA-Seq analysis revealed increased intron retention rates. Surprisingly, we identified in a screen for Num1 interacting proteins not only NTC core components as Prp19 and Cef1, but several proteins with putative functions during vesicle-mediated transport processes. Among others, Num1 interacts with the motor protein Kin1 in the cytoplasm. Similar phenotypes with respect to filamentous and polar growth, vacuolar morphology, as well as the motility of early endosomes corroborate the genetic interaction between Num1 and Kin1. Our data implicate a previously unidentified connection between a component of the splicing machinery and cytoplasmic transport processes. As the num1 deletion also affects cytoplasmic mRNA transport, the protein may constitute a novel functional interconnection between the two disparate processes of splicing and trafficking.

Indexed as

Alternative SplicingBiological TransportCarrier ProteinsCell Cycle CheckpointsCell Cycle ProteinsCytoskeletal ProteinsDNA DamageFungal ProteinsHyphaeKinesinsMolecular Sequence DataMutationRNA-Binding ProteinsSaccharomyces cerevisiae ProteinsSpliceosomesUstilagoCarrier ProteinsCell Cycle ProteinsCytoskeletal ProteinsFungal ProteinsKIN1 protein, Ustilago maydisKinesinsNUM1 protein, S cerevisiaeRNA-Binding ProteinsSaccharomyces cerevisiae Proteins

Identifiers

PMID24391515
PMCPMC3879195
OpenAlexW2001196665

What OpenQuestion holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.